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Quinolinate

 
National Institutes of Health

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Highly Cited
2008
Highly Cited
2008
Leaf senescence in Arabidopsis thaliana is a strict, genetically controlled nutrient recovery program, which typically progresses… Expand
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Highly Cited
2006
Highly Cited
2006
The brain levels of the endogenous excitotoxin quinolinic acid (QUIN) and its bioprecursor, the free radical generator 3… Expand
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Highly Cited
2004
Highly Cited
2004
Huntington's disease (HD), an inherited neurodegenerative disorder, is caused by an abnormal polyglutamine expansion in the… Expand
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Highly Cited
2003
Highly Cited
2003
Previous studies have demonstrated two different biosynthetic pathways to quinolinate, the universal de novo precursor to the… Expand
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Highly Cited
2002
Highly Cited
2002
Abstract.Rationale: Lesions of the orbital prefrontal cortex (OPFC) can cause pathologically impulsive behaviour in humans. Inter… Expand
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Highly Cited
2002
Highly Cited
2002
Previous work suggests N-methyl-D-aspartate receptor (NMDAR) activation may be involved in degeneration of medium-sized spiny… Expand
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Highly Cited
2001
Highly Cited
2001
BACKGROUND Metabolites of the kynurenine pathway of tryptophan degradation may play a role in the pathogenesis of several human… Expand
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Highly Cited
1999
Highly Cited
1999
L-3-Hydroxykynurenine (L-3-HK) and quinolinate (QUIN) are two metabolites of the kynurenine pathway, the major route of… Expand
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Review
1993
Review
1993
In a little more than 10 years, the kynurenine metabolites of tryptophan have emerged from their former position as biochemical… Expand
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Highly Cited
1991
Highly Cited
1991
The effects on the expression of a conditioned place preference of bilateral, excitotoxic amino acid-induced lesions of the… Expand
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