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Bradykinin and inflammatory pain

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An iontophoretic investigation of the actions of convulsant kynurenines and their interaction with the endogenous excitant quinolinic acid

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Antihyperalgesic effects of delta opioid agonists in a rat model of chronic inflammation.

The results indicate that mu and delta opioid receptors in the brain modulate descending pain pathways to attenuate the nociceptive response to acute thermal stimuli in both normal and inflamed tissues.
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Quinolinic acid: a potent endogenous excitant at amino acid receptors in CNS.

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Kinins and kinin receptors in the nervous system

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Activation of brown adipose tissue thermogenesis by the ventromedial hypothalamus

It is found that electrical stimulation of this area produced increased BAT thermogenesis, which suggests that the VMH exerts a dual influence in the regulation of energy balanceā€”an inhibitory effect on energy intake and a stimulatory effect on thermogenesis and energy output.
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Pro-nociceptive effects of neuromedin u in rat

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Antinociceptive activity of the bradykinin B1 and B2 receptor antagonists, des-Arg9, [Leu8]-BK and HOE 140, in two models of persistent hyperalgesia in the rat

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Antihyperalgesic effects of Ī“ opioid agonists in a rat model of chronic inflammation

The results indicate that Ī¼ and Ī“ opioid receptors in the brain modulate descending pain pathways to attenuate the nociceptive response to acute thermal stimuli in both normal and inflamed tissues.
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The involvement of bradykinin B1 and B2 receptor mechanisms in cytokineā€induced mechanical hyperalgesia in the rat

The data suggest that both bradykinin B1 and B2 receptors are involved in the induction and maintenance of cytokineā€induced hyperalgesia and show that the induction of B1 receptorā€mediated hyperAlgesia requires both cycloā€oxygenase products and ILā€1 in vivo.
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