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X-Box Binding Protein 1, Human

Known as: Tax-Responsive Element-Binding Protein 5, TREB5 protein, human, tax-responsive element-binding protein 5, human 
X-box binding protein 1 (261 aa, ~29 kDa) is encoded by the human XBP1 gene. This protein is a bZIP transcription factor that is involved in both the… Expand
National Institutes of Health

Papers overview

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Highly Cited
2014
Highly Cited
2014
Activation of the ER stress response is associated with malignant progression of B cell chronic lymphocytic leukemia (CLL). We… Expand
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Highly Cited
2009
Highly Cited
2009
Mutations in superoxide dismutase-1 (SOD1) cause familial amyotrophic lateral sclerosis (fALS). Recent evidence implicates… Expand
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Highly Cited
2008
Highly Cited
2008
The unfolded protein response (UPR) is a conserved adaptive reaction that increases cell survival under endoplasmic reticulum (ER… Expand
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Highly Cited
2005
Highly Cited
2005
The secretory function of cells relies on the capacity of the endoplasmic reticulum (ER) to fold and modify nascent polypeptides… Expand
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Highly Cited
2003
Highly Cited
2003
The mammalian unfolded protein response (UPR) protects the cell against the stress of misfolded proteins in the endoplasmic… Expand
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Highly Cited
2003
Highly Cited
2003
The transcription factor X-box binding protein 1 (XBP-1) is essential for the differentiation of plasma cells and the unfolded… Expand
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Review
2003
Review
2003
X-box binding protein-1 (XBP-1) is a transcription factor essential for plasma cell differentiation. XBP-1 transcripts are found… Expand
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Highly Cited
2002
Highly Cited
2002
The unfolded protein response (UPR), caused by stress, matches the folding capacity of endoplasmic reticulum (ER) to the load of… Expand
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Highly Cited
2001
Highly Cited
2001
Considerable progress has been made in identifying the transcription factors involved in the early specification of the B… Expand
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Highly Cited
2000
Highly Cited
2000
Transcription of genes encoding molecular chaperones and folding enzymes in the endoplasmic reticulum (ER) is induced by… Expand
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