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DL 111-IT

Known as: DL111-IT 
 
National Institutes of Health

Papers overview

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2005
2005
AIM To evaluate the antiproliferative activity of contragestazol (DL111-IT) on the human prostate cancer cell line PC3 in vitro… Expand
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2003
2003
This study evaluated the effectiveness and acute toxicity of DL111-IT combined with mifepristone (RU486) and misoprostol (MISO… Expand
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2003
2003
An analytical method was developed for determining diphenytriazol and its related impurities in oil injection by using RP-HPLC… Expand
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2000
2000
The effectiveness of DL111-IT and RU486 given alone and in combination for terminating early pregnancy was tested in the rat… Expand
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2000
2000
AIM To observe the effects of DL111 IT on decidual cells, content of PGE 2 and PGF 2α , and uterine contractibility of pregnant… Expand
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1999
1999
The present studies were conducted to evaluate the effects of DL111-IT [3-(2-ethyl phenyl)-5-(3-methoxy phenyl)-1H-1,2,4 triazol… Expand
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1997
1997
AIM To study the influence of DL111-IT on progesterone biosynthesis of cultured luteal cells (LC). METHODS LC viability was… Expand
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1997
1997
目的:研究抗孕唑(DL111IT)对离体大鼠黄体细胞孕酮合成和存活率的影响.方法:体外培养大鼠黄体细胞,用细胞计数和放射免疫法.结果:DL111IT使黄体细胞存活率随剂量上升而下降,ED50为77(71-85)mg·L-1.DL111IT3mg·L-1使黄体细胞基础黄体酮分泌下降25%,并可显著抑制弗司扣林(cAMP激动剂)10μmol·L-1和孕烯醇酮(可被3βHSD催化成黄体酮)10μmol·L-1的促孕酮分泌作用,抑制率分别为43%和155%,但不抑制hCG10IU·L-1的促黄体酮分泌作用.结论:DL111IT抗早孕作用是在cAMP和3βHSD水平上影响基础黄体酮分泌功能,并于高剂量降低黄体细胞成活率. 
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1996
1996
AIM To clarify the role of DL111-IT when combined with mifepristone (Mif) on termination of early pregnancy. METHODS… Expand
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1985
1985
The immunosuppressive properties of the non-hormonal contragestional agent 3-(2-ethylphenyl)-5-(3-methoxyphenyl)-1H-1,2,4… Expand
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