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quinaprilat

Known as: (3S)-2-((S)-N-((S)-1-Carboxy-3-Phenylpropyl)Alanyl)-1,2,3,4-Tetrahydro-3-Isoquinolinecarboxylic Acid, 2-(2-((1-carboxy-3-phenylpropyl)amino)-1-oxopropyl)-1,2,3,4-tetrahydro-3-isoquinolinecarboxylic acid, 3-Isoquinolinecarboxylic Acid, 2-(2-((1-Carboxy-3-Phenylpropyl)Amino)-1-Oxopropyl)-1,2,3,4-Tetrahydro-, (3S-(2(R*(R*)),3R*))- 
 
National Institutes of Health

Papers overview

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2002
2002
Background Angiotensin‐converting enzyme inhibitors may affect reactive oxygen species in humans in vitro and in vivo. In the… Expand
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2002
2002
A high-performance liquid chromatography (HPLC) procedure for the simultaneous determination of quinapril and its active… Expand
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Highly Cited
2001
Highly Cited
2001
Abstract—ACE inhibitors block B2 receptor desensitization, thereby potentiating bradykinin beyond blocking its hydrolysis… Expand
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Highly Cited
1999
Highly Cited
1999
BACKGROUND Endothelial cells (ECs) represent the critical cellular element responsible for postnatal angiogenesis. Because ACE… Expand
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Highly Cited
1998
Highly Cited
1998
BACKGROUND Chronic heart failure (CHF) is associated with endothelial dysfunction, including impaired flow-dependent (endothelium… Expand
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Highly Cited
1997
Highly Cited
1997
BACKGROUND The angiotensin-converting enzyme (ACE) not only generates angiotensin II but is also the main enzyme that destroys… Expand
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1997
1997
The beneficial therapeutic effects of angiotensin-converting enzyme (ACE) inhibitors are the result of reduced angiotensin II… Expand
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Highly Cited
1996
Highly Cited
1996
Mesangial cell growth and accumulation of extracellular matrix proteins constitute key features of progressive glomerular injury… Expand
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1995
1995
Quinapril and quinaprilat concentrations were determined in perfusate, urine, and perfusate ultrafiltrate using a specific and… Expand
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Highly Cited
1994
Highly Cited
1994
The angiotensin I (AI) metabolite, A(1–7), elicited a concentration‐dependent dilator response (ED50 ≥ 2 μm) in porcine coronary… Expand
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