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mitochondria-associated protein catabolic process
Known as:
mitochondrion-associated protein catabolic process
, mitochondria-associated protein degradation
, MAD
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The chemical reactions and pathways resulting in the breakdown of proteins transported from mitochondria and targeted to cytoplasmic proteasomes for…
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National Institutes of Health
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Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
2009
2009
Mad Is Required for Wingless Signaling in Wing Development and Segment Patterning in Drosophila
Edward Eivers
,
L. Fuentealba
,
Veronika Sander
,
J. C. Clemens
,
L. Hartnett
,
E. D. De Robertis
PLoS ONE
2009
Corpus ID: 3776583
A key question in developmental biology is how growth factor signals are integrated to generate pattern. In this study we…
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Highly Cited
2003
Highly Cited
2003
dSmurf Selectively Degrades Decapentaplegic-activated MAD, and Its Overexpression Disrupts Imaginal Disc Development*
Yao-Yun Liang
,
Xia Lin
,
+5 authors
Xin-Hua Feng
Journal of Biological Chemistry
2003
Corpus ID: 24710638
MAD plays an important role in decapentaplegic (DPP) signaling throughout Drosophila development. Despite a recent study…
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Review
2001
Review
2001
The interplay between Mad and Myc in proliferation and differentiation.
Z. Q. Zhou
,
P. Hurlin
Trends in Cell Biology
2001
Corpus ID: 39280912
Highly Cited
1997
Highly Cited
1997
The MAD-related protein Smad7 associates with the TGFbeta receptor and functions as an antagonist of TGFbeta signaling.
H. Hayashi
,
S. Abdollah
,
+8 authors
D. Falb
Cell
1997
Corpus ID: 44998203
TGFbeta signaling is initiated when the type I receptor phosphorylates the MAD-related protein, Smad2, on C-terminal serine…
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Highly Cited
1997
Highly Cited
1997
Vascular MADs: two novel MAD-related genes selectively inducible by flow in human vascular endothelium.
J. N. Topper
,
Jiexing Cai
,
+12 authors
D. Falb
Proceedings of the National Academy of Sciences…
1997
Corpus ID: 26099605
Vascular endothelium is an important transducer and integrator of both humoral and biomechanical stimuli within the…
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Highly Cited
1995
Highly Cited
1995
Effects of the MYC oncogene antagonist, MAD, on proliferation, cell cycling and the malignant phenotype of human brain tumour cells
Jun Chen
,
T. Willingham
,
L. Margraf
,
N. Schreiber-Agus
,
R. DePinho
,
P. Nisen
Nature Network Boston
1995
Corpus ID: 23968945
To investigate how overexpression of MAD, an antagonist of MYC oncogenes influences the malignant phenotype of human cancer cells…
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Highly Cited
1995
Highly Cited
1995
Differential effects by Mad and Max on transformation by cellular and viral oncoproteins.
C. Cerni
,
K. Bousset
,
C. Seelos
,
H. Burkhardt
,
M. Henriksson
,
B. Lüscher
Oncogene
1995
Corpus ID: 24964691
c-Myc is an essential component of the regulatory mechanisms controlling cell growth. Max is the obligatory partner of c-Myc for…
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Highly Cited
1994
Highly Cited
1994
Suppression of Myc, but not E1a, transformation activity by Max-associated proteins, Mad and Mxi1.
Enrique Gómez Lahoz
,
Lin Xu
,
N. Schreiber-Agus
,
R. DePinho
Proceedings of the National Academy of Sciences…
1994
Corpus ID: 24893924
Mad and Mxi1, two members of the Myc-related basic-region helix-loop-helix/leucine-zipper family of proteins, associate directly…
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Highly Cited
1994
Highly Cited
1994
Mapping of two genes encoding members of a distinct subfamily of MAX interacting proteins: MAD to human chromosome 2 and mouse chromosome 6, and MXI1 to human chromosome 10 and mouse chromosome 19.
S. Edelhoff
,
D. Ayer
,
+6 authors
C. Disteche
Oncogene
1994
Corpus ID: 25024494
Both the MAD and the MXI1 genes encode basic-helix-loop-helix-leucine zipper (bHLH-Zip) transcription factors which bind Max in…
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Highly Cited
1986
Highly Cited
1986
The multiple acyl-coenzyme A dehydrogenation disorders, glutaric aciduria type II and ethylmalonic-adipic aciduria. Mitochondrial fatty acid oxidation, acyl-coenzyme A dehydrogenase, and electron…
B. Amendt
,
W. Rhead
Journal of Clinical Investigation
1986
Corpus ID: 6862453
The multiple acyl-coenzyme A (CoA) dehydrogenation disorders (MAD) include severe (S) and mild (M) variants, glutaric aciduria…
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