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micacocidin A

Known as: micacocidin-A, micacocidin-A, Zn(2+) 
 
National Institutes of Health

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Highly Cited
2010
Highly Cited
2010
Although a key factor in Alzheimer's disease etiology is enrichment of Zn(2+) in aggregates, and there are data suggesting that… Expand
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Highly Cited
2005
Highly Cited
2005
Transient global or forebrain ischemia induced experimentally in animals can cause selective, delayed neuronal death of… Expand
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Review
2004
Review
2004
The role of Zn2+ in the CNS has remained enigmatic for several decades. This divalent cation is accumulated by specific neurons… Expand
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Highly Cited
2003
Highly Cited
2003
Recent evidence suggests that intracellular Zn(2+) accumulation contributes to the neuronal injury that occurs in epilepsy or… Expand
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Highly Cited
2002
Highly Cited
2002
The expression of mRNA for acid sensing ion channels (ASIC) subunits ASIC1a, ASIC2a and ASIC2b has been reported in hippocampal… Expand
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Highly Cited
2001
Highly Cited
2001
Rapid entry of Ca(2+) or Zn(2+) kills neurons. Mitochondria are major sites of Ca(2+)-dependent toxicity. This study examines Zn… Expand
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Highly Cited
2001
Highly Cited
2001
Zn(2+) is found in glutamatergic nerve terminals throughout the mammalian forebrain and has diverse extracellular and… Expand
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Highly Cited
2001
Highly Cited
2001
The mammalian CNS contains an abundance of chelatable Zn(2+) sequestered in the vesicles of glutamatergic terminals. These… Expand
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Highly Cited
2001
Highly Cited
2001
Two new fluorescent sensors for Zn(2+) that utilize fluorescein as a reporting group, Zinpyr-1 and Zinpyr-2, have been… Expand
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Highly Cited
2001
Highly Cited
2001
In mitosis, the anaphase-promoting complex (APC) regulates the onset of sister-chromatid separation and exit from mitosis by… Expand
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