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Calcium Channel Agonists

Known as: Channel Agonists, Calcium, Channel Activators, Calcium, Agonists, Calcium Channel 
Agents that increase calcium influx into calcium channels of excitable tissues. This causes vasoconstriction in VASCULAR SMOOTH MUSCLE and/or CARDIAC… 
National Institutes of Health

Related topics

Related topics

13 relations

Narrower (6)

1-ethyl-2-benzimidazolinoneBay-K-8644CGP 28392Calcitriol
Calcium ChannelIn Bloodaspects of radiation effectschemical synthesis

Broader (2)

Cardiovascular AgentsVasoconstrictor Agents

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
Highly Cited
2002
Highly Cited
2002
Development of a 'mechano-active' scaffold for tissue engineering.
Highly Cited
1995
Highly Cited
1995
The steroidogenic acute regulatory protein is induced by angiotensin II and K+ in H295R adrenocortical cells
Highly Cited
1989
Highly Cited
1989
Voltage-sensitive calcium flux into bovine chromaffin cells occurs through dihydropyridine-sensitive and dihydropyridine- and ω-conotoxin-insensitive pathways
Highly Cited
1988
Highly Cited
1988
Anticonvulsant profile of the dihydropyridine calcium channel antagonists, nitrendipine and nimodipine.
Highly Cited
1987
Highly Cited
1987
Differential effects of calcium entry blockers on pre- and postsynaptic influx of calcium in the rat hippocampus in vitro
1986
1986
Selective enhancement of angiotensin II- and potassium-stimulated aldosterone secretion by the calcium channel agonist BAY K 8644.
Recent studies with dihydropyridine calcium channel antagonists have indicated that voltage-sensitive calcium channels (VSCC… 
1986
1986
Control of PTH secretion is mediated through calcium channels and is blocked by pertussis toxin treatment of parathyroid cells.
Highly Cited
1985
Highly Cited
1985
Dihydropyridine calcium channel activators and antagonists influence depolarization-evoked inositol phospholipid hydrolysis in brain.
Review
1985
Review
1985
Modulation of calcium channel function by drugs.
Highly Cited
1985
Highly Cited
1985
Characteristics of angiotensin II-, K+- and ACTH-induced calcium influx in adrenal glomerulosa cells. Evidence that angiotensin II, K+, and ACTH may open a common calcium channel.
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