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arachidonoylserotonin

Known as: AA-5-HT 
National Institutes of Health

Related topics

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3 relations

Broader (2)

Arachidonic AcidsSerotonin
analogs & derivatives

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
Highly Cited
2011
Highly Cited
2011
The blockade of the transient receptor potential vanilloid type 1 and fatty acid amide hydrolase decreases symptoms and central sequelae in the medial prefrontal cortex of neuropathic rats
BackgroundNeuropathic pain is a chronic disease resulting from dysfunction within the "pain matrix". The basolateral amygdala… 
Highly Cited
2011
Highly Cited
2011
Presence, formation and putative biological activities of N-acyl serotonins, a novel class of fatty-acid derived mediators, in the intestinal tract.
Highly Cited
2009
Highly Cited
2009
Anxiolytic Effects in Mice of a Dual Blocker of Fatty Acid Amide Hydrolase and Transient Receptor Potential Vanilloid Type-1 Channels
The endocannabinoid-inactivating enzyme, fatty acid amide hydrolase (FAAH), and the transient receptor potential vanilloid type-1… 
Review
2008
Review
2008
Overview of the chemical families of fatty acid amide hydrolase and monoacylglycerol lipase inhibitors.
The family of the endogenous agonists of the cannabinoid receptors--i.e., the endocannabinoids--includes several polyunsaturated… 
Highly Cited
2008
Highly Cited
2008
The role of endocannabinoids in the regulation of gastric emptying: alterations in mice fed a high‐fat diet
Endocannabinoids (via cannabinoid CB1 receptor activation) are physiological regulators of intestinal motility and food intake… 
Highly Cited
2008
Highly Cited
2008
The analgesic effect of N-arachidonoyl-serotonin, a FAAH inhibitor and TRPV1 receptor antagonist, associated with changes in rostral ventromedial medulla and locus coeruleus cell activity in rats
Highly Cited
2007
Highly Cited
2007
Analgesic actions of N‐arachidonoyl‐serotonin, a fatty acid amide hydrolase inhibitor with antagonistic activity at vanilloid TRPV1 receptors
N‐arachidonoyl‐serotonin (AA‐5‐HT) is an inhibitor of fatty acid amide hydrolase (FAAH)‐catalysed hydrolysis of the… 
Highly Cited
2005
Highly Cited
2005
Inhibition of fatty-acid amide hydrolase enhances cannabinoid stress-induced analgesia: Sites of action in the dorsolateral periaqueductal gray and rostral ventromedial medulla
Highly Cited
2004
Highly Cited
2004
A new strategy to block tumor growth by inhibiting endocannabinoid inactivation
Endocannabinoid signaling has been shown to be enhanced in several cancer tissues and malignant cells, and studies in cell lines… 
Highly Cited
1998
Highly Cited
1998
Arachidonoylserotonin and other novel inhibitors of fatty acid amide hydrolase.
Fatty acid amide hydrolase (FAAH) catalyzes the hydrolysis of bioactive fatty acid amides and esters such as the endogenous… 
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