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HOE 694

Known as: HOE-694, HOE694 
 
National Institutes of Health

Papers overview

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Highly Cited
1999
Highly Cited
1999
OBJECTIVE Fluid shear stress (the frictional force resulting from blood flow) is a principal regulator of endothelial nitric… Expand
Highly Cited
1999
Highly Cited
1999
1 Receptor‐mediated endocytosis is an important mechanism for transport of macromolecules and regulation of cell‐surface receptor… Expand
Highly Cited
1997
Highly Cited
1997
Chronic metabolic acidosis (CMA) is associated with an adaptive increase in the bicarbonate absorptive capacity of the rat… Expand
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Highly Cited
1996
Highly Cited
1996
The aims of the present study were to estimate the fraction of renal brush border membrane Na+-H+ exchange activity mediated by… Expand
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Highly Cited
1996
Highly Cited
1996
Na+/H+ exchangers (NHEs) are transporters that exchange sodium and proton ions across the plasma membrane at the expense of their… Expand
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Review
1996
Review
1996
During myocardial ischemia, a large reduction of tissue pH develops, and tissue pH returns to normal after reperfusion. In recent… Expand
Highly Cited
1995
Highly Cited
1995
OBJECTIVE Stimulation of myocardial alpha 1 adrenoceptors causes (1) exacerbation of reperfusion induced arrhythmias, and (2… Expand
Highly Cited
1994
Highly Cited
1994
BACKGROUND Experiments were carried out using the new Na(+)-H+ exchange inhibitor (3-methylsulfonyl-4-piperidinobenzoyl)guanidine… Expand
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Highly Cited
1993
Highly Cited
1993
A fibroblast mutant cell line devoid of Na+/H+ exchange was used to stably express cDNAs encoding the NHE1, NHE2, and NHE3 Na+/H… Expand
Highly Cited
1993
Highly Cited
1993
1 The benzoylguanidine derivative Hoe 694 ((3‐methylsulphonyl‐4‐piperidino‐benzoyl) guanidine methanesulphonate) was… Expand