Skip to search form
Skip to main content
Skip to account menu
Semantic Scholar
Semantic Scholar's Logo
Search 210,256,672 papers from all fields of science
Search
Sign In
Create Free Account
CD5 gene
Known as:
CD5 molecule
, Ly1, MOUSE, HOMOLOG OF
, T1
Expand
This gene may be involved in the proliferation of T-cells.
National Institutes of Health
Create Alert
Alert
Related topics
Related topics
5 relations
Antigens, CD5
Ligand Binding
T-Cell Proliferation
T-Cell Surface Glycoprotein CD5, human
Narrower (1)
CD5 wt Allele
Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
2014
2014
Interaction of CD5 and CD72 is involved in regulatory T and B cell homeostasis
M. Zheng
,
C. Xing
,
+8 authors
R. Wang
Immunological Investigations
2014
Corpus ID: 40416033
Regulatory IL-10-producing CD1dhighCD5+CD19+ B cells and CD4+CD25+Foxp3+ T cells have been found to modulate immune responses in…
Expand
2004
2004
KappaM-conotoxin RIIIK, structural and functional novelty in a K+ channel antagonist.
A. Al-Sabi
,
D. Lennartz
,
+5 authors
H. Terlau
Biochemistry
2004
Corpus ID: 25947694
Venomous organisms have evolved a variety of structurally diverse peptide neurotoxins that target ion channels. Despite the lack…
Expand
2002
2002
Deletion analysis of chromosome 13q14.3 and characterisation of an alternative splice form of LEU1 in B cell chronic lymphocytic leukemia
C. Rowntree
,
V. Duke
,
+4 authors
L. Foroni
Leukemia
2002
Corpus ID: 853676
Heterozygous and homozygous deletions of chromosome 13q14.3 are found in 50% of patients with B cell CLL, suggesting the presence…
Expand
2000
2000
Comparative sequence analysis of a region on human chromosome 13q14, frequently deleted in B-cell chronic lymphocytic leukemia, and its homologous region on mouse chromosome 14.
B. Kapanadze
,
N. Makeeva
,
+13 authors
O. Sangfelt
Genomics
2000
Corpus ID: 9712293
Previous studies have indicated the presence of a putative tumor suppressor gene on human chromosome 13q14, commonly deleted in…
Expand
Highly Cited
1997
Highly Cited
1997
Cloning of two candidate tumor suppressor genes within a 10 kb region on chromosome 13q14, frequently deleted in chronic lymphocytic leukemia
Yie Liu
,
M. Corcoran
,
+21 authors
D. Oscier
Oncogene
1997
Corpus ID: 21133945
Previous studies have indicated the presence of a putative tumor suppressor gene on chromosome 13q14, commonly deleted in…
Expand
Highly Cited
1994
Highly Cited
1994
Efficient targeted integration at leu1-32 and ura4-294 in Schizosaccharomyces pombe.
J. Keeney
,
J. Boeke
Genetics
1994
Corpus ID: 28000728
Homologous integration into the fission yeast Schizosaccharomyces pombe has not been well characterized. In this study, we have…
Expand
Highly Cited
1992
Highly Cited
1992
Intracellular events involved in CD5-induced human T cell activation and proliferation.
J. Alberola-Ila
,
L. Places
,
D. Cantrell
,
J. Vives
,
F. Lozano
Journal of Immunology
1992
Corpus ID: 22649324
In this report we describe a novel pathway of human T cell activation and proliferation involving the CD5 surface Ag. The CD5…
Expand
Highly Cited
1986
Highly Cited
1986
Phenotypic and Functional Characterization of Human LEUI (CD5) B Cells
N. Gadol
,
K. Ault
Immunological Reviews
1986
Corpus ID: 24628176
Leu1 B cells have been observed in high numbers in CLL patients, fetal spleen, and in bone marrow transplant recipients. We…
Expand
1986
1986
Epitope Expression on Primate Lymphocyte Surface Antigens
Y. Murayama
,
K. Fukao
,
A. Noguchi
,
O. Takenaka
Journal of medical primatology
1986
Corpus ID: 6046507
The cross‐reactivity of peripheral blood mononuclear cells from 28 nonhuman primates was investigated with ten kinds of Leu…
Expand
Highly Cited
1984
Highly Cited
1984
Yeast LEU1. Repression of mRNA levels by leucine and relationship of 5'-noncoding region to that of LEU2.
Y. Hsu
,
P. Schimmel
Journal of Biological Chemistry
1984
Corpus ID: 32285196
By clicking accept or continuing to use the site, you agree to the terms outlined in our
Privacy Policy
(opens in a new tab)
,
Terms of Service
(opens in a new tab)
, and
Dataset License
(opens in a new tab)
ACCEPT & CONTINUE