BRL-38226

Known as: BRL 38226, BRL38226 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

1990-1998
012319901998

Papers overview

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1998
1998
This study examined the pharmacological characteristics of binding sites for the potent K+ channel opener [3H]P1075, as well as… (More)
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1994
1994
The effects of racemic cromakalim and the (+)-3R,4S and (-)-3S,4R isomers, BRL 38226 and levcromakalim respectively, on the… (More)
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1993
1993
Rat isolated tracheae were labelled by incubation with [3H]choline to measure the tritium efflux elicited by electrical… (More)
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1992
1992
1. In guinea-pig isolated bronchus treated with indomethacin (2.8 microM), electrical field stimulation (EFS; 10 Hz, 0.5 ms, 60… (More)
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1992
1992
Binding of [125I]-labelled endothelin-1 (ET-1) to rat cardiac membranes and the effects of endothelin-1 (ET-1), endothelin-3 (ET… (More)
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1992
1992
The effects of cromakalim and its active enantiomer BRL 38226 on voltage-gated Ca2+ channels in smooth muscle cells isolated from… (More)
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1991
1991
K+ channel openers elicit K+ currents in follicle-enclosed Xenopus oocytes. The most potent activators are the pinacidil… (More)
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1991
1991
1. The ability of several K-channel openers to inhibit KCl-induced contractions of rat bladder detrusor and spontaneous… (More)
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1991
1991
Specific binding sites for [3H]glibenclamide, a potent ATP-sensitive K+ channel blocker, have been characterized in the isolated… (More)
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1990
1990
Potassium (K+) channels are present on airway smooth muscle cells, and their activation results in hyperpolarization and… (More)
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