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unc-78 protein, C elegans

Known as: unc78 protein, C elegans, Aip1 protein, C elegans, unc78 protein, Caenorhabditis elegans 
 
National Institutes of Health

Related topics

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2 relations

Broader (2)

Caenorhabditis elegans ProteinsMicrofilament Proteins

Papers overview

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2020
2020
Caenorhabditis elegans AF4/FMR2 Family Homolog affl-2 Regulates Heat-Shock-Induced Gene Expression
To mitigate the deleterious effects of temperature increases on cellular organization and proteotoxicity, organisms have… Expand
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2019
2019
Single-Copy Knock-In Loci for Defined Gene Expression in Caenorhabditis elegans
We have generated a single-copy knock-in loci for defined gene expression (SKI LODGE) system to insert any DNA by CRISPR/Cas9 at… Expand
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2017
2017
Knock-out of a mitochondrial sirtuin protects neurons from degeneration in Caenorhabditis elegans
Sirtuins are NAD⁺-dependent deacetylases, lipoamidases, and ADP-ribosyltransferases that link cellular metabolism to multiple… Expand
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2016
2016
Activation of the Caenorhabditis elegans Degenerin Channel by Shear Stress Requires the MEC-10 Subunit*
Mechanotransduction in Caenorhabditis elegans touch receptor neurons is mediated by an ion channel formed by MEC-4, MEC-10, and… Expand
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Highly Cited
2012
Highly Cited
2012
ALS mutations in FUS cause neuronal dysfunction and death in Caenorhabditis elegans by a dominant gain-of-function mechanism
It is unclear whether mutations in fused in sarcoma (FUS) cause familial amyotrophic lateral sclerosis via a loss-of-function… Expand
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Highly Cited
2003
Highly Cited
2003
Dopaminergic neuronal loss and motor deficits in Caenorhabditis elegans overexpressing human α‐synuclein
Overexpression of human α‐synuclein in model systems, including cultured neurons, drosophila and mice, leads to biochemical and… Expand
Highly Cited
2003
Highly Cited
2003
Loss of kindlin-1, a human homolog of the Caenorhabditis elegans actin-extracellular-matrix linker protein UNC-112, causes Kindler syndrome.
Kindler syndrome is an autosomal recessive disorder characterized by neonatal blistering, sun sensitivity, atrophy, abnormal… Expand
Highly Cited
2003
Highly Cited
2003
Neurodegeneration and defective neurotransmission in a Caenorhabditis elegans model of tauopathy
Frontotemporal dementia with parkinsonism chromosome 17 type (FTDP-17) is caused by mutations in MAPT, the gene encoding tau… Expand
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Highly Cited
1993
Highly Cited
1993
Oxidative stress and ageing in Caenorhabditis elegans.
Mutations in the age-1 gene double both the mean and maximum life span of Caenorhabditis elegans. They also result in an age… Expand
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Review
1989
Review
1989
Genetic control of differentiation of the Caenorhabditis elegans touch receptor neurons.
The genetic control of neuronal differentiation has been studied by examining mutations that affect the development and function… Expand
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