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concanamycin A

Known as: folimycin 
 
National Institutes of Health

Papers overview

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Highly Cited
2010
Highly Cited
2010
Key components of vesicular neurotransmitter release, such as Ca(2+) influx and membrane recycling, are affected by cytosolic pH… Expand
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Highly Cited
2007
Highly Cited
2007
Recently, it has been claimed that vesicles recycling spontaneously and during activity belong to different pools. Here we… Expand
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Highly Cited
2004
Highly Cited
2004
Botulinum neurotoxins (BoNTs) act within the synaptic terminal to block neurotransmitter release. The toxin enters the neuron by… Expand
Highly Cited
1997
Highly Cited
1997
T cell activation by specific antigen results in a rapid and long-lasting downregulation of triggered T cell receptors (TCRs). In… Expand
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Highly Cited
1996
Highly Cited
1996
Perforin- and Fas-based cytolytic pathways are two major mechanisms of cell-mediated cytotoxicity. Recently, we have shown that… Expand
Highly Cited
1996
Highly Cited
1996
Heavy endosomes were isolated from proximal tubules using a combination of magnesium precipitation and wheat-germ agglutinin… Expand
Highly Cited
1994
Highly Cited
1994
Destruxin B, a peptide antibiotic, inhibits vacuolar-type ATPase (V-ATPase) specifically and dose-dependently among ATPases… Expand
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Highly Cited
1994
Highly Cited
1994
An inhibitor of vacuolar type H(+)-ATPase, concanamycin A (CMA), inhibited the specific cytolytic activity of a CD8+ CTL clone… Expand
Highly Cited
1993
Highly Cited
1993
Folimycin (concanamycin A) specifically inhibited vacuolar-type ATPase as far as examined. Folimycin blocked excretion of the… Expand
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Highly Cited
1984
Highly Cited
1984
Concanamycins A, B and C were isolated from the mycelium of Streptomyces diastatochromogenes S-45 as effective inhibitors of the… Expand
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