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camostat

Known as: N,N-dimethylcarbamoylmethyl-4-(4-guanidinobenzoyloxy)phenylacetate methanesulfonate, camostate 
 
National Institutes of Health

Papers overview

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Highly Cited
2009
Highly Cited
2009
Inhibition of airway epithelial sodium channel (ENaC) function enhances mucociliary clearance (MCC). ENaC is positively regulated… Expand
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Highly Cited
1996
Highly Cited
1996
The rat pancreatic carcinoma cell line AR4-2J was screened for growth-associated genes linked to the mitogenic effect of the… Expand
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1990
1990
The factors operating at the apical side of the endocrine cell releasing cholecystokinin (CCK) were investigated using the… Expand
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Highly Cited
1989
Highly Cited
1989
The present study evaluates the effect of atropine and of the cholecystokinin receptor antagonist loxiglumide on feedback… Expand
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Highly Cited
1988
Highly Cited
1988
Chronic feeding of rats with camostate results in pancreatic hypertrophy or hyperplasia, or both. Previous studies suggest that… Expand
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Highly Cited
1988
Highly Cited
1988
Abstract. Pancreatic secretion and plasma cholecystokinin (CCK) and secretin levels were measured in 10 healthy volunteers after… Expand
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Highly Cited
1987
Highly Cited
1987
The effects on pancreatic growth and plasma CCK concentration of chronic feeding of camostate (400 mg/kg day for 10 days), a… Expand
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Highly Cited
1987
Highly Cited
1987
Pancreatic exocrine function in rats given synthetic protease inhibitor camostate (200 mg/kg body weight) perorally once daily… Expand
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Highly Cited
1986
Highly Cited
1986
The serine proteinase inhibitor camostate was fed to rats in single, daily doses of 100 mg/kg, 200 mglkg, and 400 mg/kg for 5, 10… Expand
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1984
1984
Camostate, a synthetic serine protease inhibitor, specifically inhibits the trypsin activity in vitro. Immediately after… Expand
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