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bifemelane

Known as: 1-Butanamine, N-methyl-4-(2-(phenylmethyl)phenoxy)-, hydrochloride, 2-(4-methylaminobutoxy)diphenylmethane, 4-(2-benzylphenoxy)-N-methylbutylamine 
National Institutes of Health

Related topics

Related topics

5 relations

Broader (2)

Antidepressive AgentsBenzhydryl Compounds

Narrower (3)

CeleportMCI 2016bifemelane hydrochloride

Papers overview

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1998
1998
Bifemelane protects cultured cortical neurons against N-methyl-D-aspartate receptor-mediated glutamate cytotoxicity.
This study was performed to examine the neuroprotective action of bifemelane against glutamate cytotoxicity in cultured cortical… 
1997
1997
New BRCA2 mutation in an Ashkenazi Jewish family with breast and ovarian cancer
1996
1996
Effects of bifemelane on muscarinic receptors and choline acetyltransferase in the brains of aged rats following chronic cerebral hypoperfusion induced by permanent occlusion of bilateral carotid…
Cerebral hypoperfusion was chronically induced in aged rats via permanent bilateral occlusion of common carotid arteries (2VO… 
1994
1994
Reduced choline acetyltransferase activity and muscarinic M1 receptor levels in aged Fisher 344 rat brains did not parallel their respective mRNA levels
1993
1993
Effects of putative cognitive function-enhancing drugs and dopaminergic agents on somatostatin and neuropeptide Y in rat brain.
1991
1991
Effects of several cerebroprotective drugs on NMDA channel function: evaluation using Xenopus oocytes and [3H]MK-801 binding.
1990
1990
Different Drug-Susceptibilities of long-term potentiation in three input systems to the CA3 region of the guinea pig hippocampus in vitro
1990
1990
Bifemelane induces translocation of protein kinase C in the CA3, but not the CA1, region of guinea-pig hippocampus.
We made use of the [3H]phorbol 12,13-dibutyrate binding assay to investigate the effects of bifemelane on the subcellular… 
1989
1989
Pharmacological effects of indeloxazine, a new cerebral activator, on brain functions distinct from other cerebral metabolic enhancers
1988
1988
Different susceptibilities of long-term potentiations in CA3 and CA1 regions of guinea pig hippocampal slices to nootropic drugs
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