Skip to search formSkip to main contentSkip to account menu

beta-adrenergic receptor kinase activity

Known as: [b-adrenergic-receptor] kinase activity, beta-receptor kinase activity, beta-adrenergic receptor-specific kinase activity 
Catalysis of the reaction: ATP + beta-adrenergic receptor = ADP + phospho-beta-adrenergic receptor. [EC:2.7.11.15, MetaCyc:BETA-ADRENERGIC-RECEPTOR… 
National Institutes of Health

Related topics

Related topics

4 relations
Beta-Adrenergic Receptor Kinase 1Beta-Adrenergic Receptor Kinase 2beta-ARK 2 beta-adrenergic receptor kinase activitybetaARK1

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
Highly Cited
2013
Highly Cited
2013
The Bile Acid Receptor TGR5 Does Not Interact with β-Arrestins or Traffic to Endosomes but Transmits Sustained Signals from Plasma Membrane Rafts*
Background: The TGR5 bile acid receptor controls energy balance, inflammation, and digestion, but TGR5 signaling is poorly… 
Highly Cited
2009
Highly Cited
2009
Regulation of GPR54 signaling by GRK2 and {beta}-arrestin.
Kisspeptin and its receptor, GPR54, are major regulators of the hypothalamic-pituitary-gonadal axis as well as regulators of… 
Highly Cited
2009
Highly Cited
2009
G Protein-coupled Receptor Kinase-2 Constitutively Regulates D2 Dopamine Receptor Expression and Signaling Independently of Receptor Phosphorylation*
We investigated the regulatory effects of GRK2 on D2 dopamine receptor signaling and found that this kinase inhibits both… 
Highly Cited
2001
Highly Cited
2001
Interaction and Feedback Regulation between STK15/BTAK/Aurora-A Kinase and Protein Phosphatase 1 through Mitotic Cell Division Cycle*
STK15 is an Aurora/Ipl-1 related serine/threonine kinase that is associated with centrosomes and induces aneuploidy when… 
Highly Cited
2001
Highly Cited
2001
The Binding Site for the @ r Subunits of Heterotrimeric G Proteins on the @-Adrenergic Receptor Kinase *
The By subunits of heterotrimeric G proteins play important roles in regulating receptor-stimulated signal transduction… 
Highly Cited
1997
Highly Cited
1997
A central role for beta-arrestins and clathrin-coated vesicle-mediated endocytosis in beta2-adrenergic receptor resensitization. Differential regulation of receptor resensitization in two distinct…
G protein-coupled receptor (GPCR) sequestration to endosomes is proposed to be the mechanism by which G protein-coupled receptor… 
Highly Cited
1997
Highly Cited
1997
Phosphorylation and Desensitization of Human Endothelin A and B Receptors
Although endothelin-1 can elicit prolonged physiologic responses, accumulating evidence suggests that rapid desensitization… 
Highly Cited
1996
Highly Cited
1996
Effect of Different G Protein-coupled Receptor Kinases on Phosphorylation and Desensitization of the -Adrenergic Receptor (*)
The α-adrenergic receptor (αAR), its truncated mutant T368, different G protein-coupled receptor kinases (GRK) and arrestin… 
Highly Cited
1995
Highly Cited
1995
Reduced beta-adrenergic receptor activation decreases G-protein expression and beta-adrenergic receptor kinase activity in porcine heart.
To determine whether beta-adrenergic receptor agonist activation influences guanosine 5'-triphosphate-binding protein (G-protein… 
Highly Cited
1993
Highly Cited
1993
Identification of novel pleckstrin homology (PH) domains provides a hypothesis for PH domain function.
Pleckstrin homology (PH) domains are difficult to find in protein sequence databases with widely used computer programs. A simple… 
By clicking accept or continuing to use the site, you agree to the terms outlined in our Privacy Policy (opens in a new tab), Terms of Service (opens in a new tab), and Dataset License (opens in a new tab)