Yersinia pestis Ab:Titr:Pt:Ser:Qn

Known as: Yersinia pestis As:Titer:Moment:Serum:Kwantitatief, ????? ??? ??:??:????:??:??, anticuerpo contra Yersinia pestis:factor de dilución (titulación):punto en el tiempo:suero:cuantitativo 
 
National Institutes of Health

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AntibodiesMicrobiology procedureSerumTiter
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Highly Cited
2014
Highly Cited
2014
Caspase-8 and RIP kinases regulate bacteria-induced innate immune responses and cell death.
A number of pathogens cause host cell death upon infection, and Yersinia pestis, infamous for its role in large pandemics such as… (More)
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Highly Cited
2008
Highly Cited
2008
Caspase-1 activation in macrophages infected with Yersinia pestis KIM requires the type III secretion system effector YopJ.
Pathogenic Yersinia species utilize a type III secretion system (T3SS) to translocate effectors called Yersinia outer proteins… (More)
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2007
2007
Oral vaccination with salmonella simultaneously expressing Yersinia pestis F1 and V antigens protects against bubonic and pneumonic plague.
The gut provides a large area for immunization enabling the development of mucosal and systemic Ab responses. To test whether the… (More)
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Highly Cited
2006
Highly Cited
2006
Virulence factors of Yersinia pestis are overcome by a strong lipopolysaccharide response
At mammalian body temperature, the plague bacillus Yersinia pestis synthesizes lipopolysaccharide (LPS)–lipid A with poor Toll… (More)
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Highly Cited
2005
Highly Cited
2005
CRISPR elements in Yersinia pestis acquire new repeats by preferential uptake of bacteriophage DNA, and provide additional tools for evolutionary studies.
The remarkable repetitive elements called CRISPRs (clustered regularly interspaced short palindromic repeats) consist of repeats… (More)
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Review
2005
Review
2005
The evolution of flea-borne transmission in Yersinia pestis.
Transmission by fleabite is a recent evolutionary adaptation that distinguishes Yersinia pestis, the agent of plague, from… (More)
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Highly Cited
2005
Highly Cited
2005
Phenotypic convergence mediated by GGDEF-domain-containing proteins.
GGDEF domain-containing proteins have been implicated in bacterial signal transduction and synthesis of the second messenger… (More)
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Highly Cited
2004
Highly Cited
2004
Transmission of Yersinia pestis from an infectious biofilm in the flea vector.
Transmission of plague by fleas depends on infection of the proventricular valve in the insect's foregut by a dense aggregate of… (More)
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Highly Cited
2004
Highly Cited
2004
The plague virulence protein YopM targets the innate immune response by causing a global depletion of NK cells.
Yersinia pestis, the etiologic agent of plague, delivers six Yersinia outer proteins (Yops) into host cells upon direct bacterial… (More)
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Highly Cited
2003
Highly Cited
2003
Translocation of YopE and YopN into eukaryotic cells by Yersinia pestis yopN, tyeA, sycN, yscB and lcrG deletion mutants measured using a phosphorylatable peptide tag and phosphospecific antibodies.
Yersinia pestis, the causative agent of plague, exports a set of virulence proteins called Yops upon contact with eukaryotic… (More)
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