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R 56865

Known as: R-56865, R56865 
 
National Institutes of Health

Papers overview

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Highly Cited
2004
Highly Cited
2004
The Na+-activated K+ current was studied in inside-out patches and in whole cells isolated from the guinea-pig cardiac ventricle… Expand
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Highly Cited
2000
Highly Cited
2000
1 Persistent sodium channel activity was recorded before and during hypoxia from cell‐attached and inside‐out patches obtained… Expand
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Highly Cited
1999
Highly Cited
1999
We investigated protective effects of KB‐R7943, a Na+/Ca2+ exchange (NCX) inhibitor, on ouabain‐induced tonotropy and arrhythmias… Expand
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Highly Cited
1999
Highly Cited
1999
We hypothesized that by limiting the Na+ and Ca2+ loading by a blocker/inhibitor of the Na+ channel (lidocaine), Na+ overload… Expand
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Highly Cited
1996
Highly Cited
1996
Abstract The inhibitors of the Na+/H+-exchange (NHE1) system Hoe 694 and Hoe 642 possess cardioprotective effects in ischaemia… Expand
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Highly Cited
1995
Highly Cited
1995
Anoxic depolarization (AD) and failure of ion homeostasis play an important role in ischemia-induced neuronal injury. In the… Expand
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1995
1995
We present in this report the characteristics of the damage induced by 6-hydroxydopamine and H2O2 on bovine chromaffin cells in… Expand
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1992
1992
In a perfused guinea-pig heart model of myocardial ischaemia, reducing coronary flow by 95% for four successive 6 min periods… Expand
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Highly Cited
1991
Highly Cited
1991
Intracellular Ca2+ overload is considered to be the final pathway leading to cell death under pathological conditions. However… Expand
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1990
1990
The antiarrhythmic effects of R56865 were characterized both in vivo and in vitro. Four groups (n = 12 per group) of anesthetized… Expand
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