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Protein Fragment

A fragment of a protein, which may be a diagnostic or prognostic factor and used as a biomarker. (NCI)
National Institutes of Health

Related topics

Related topics

17 relations
Amino-terminal pro-brain natriuretic peptideBeta Isomer of C-Terminal Telopeptide of Type I CollagenC-telopeptideC-terminal type I collagen telopeptide

Papers overview

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2007
2007
X‐ray structure of the PHF core C‐terminus: Insight into the folding of the intrinsically disordered protein tau in Alzheimer's disease
2003
2003
Generation of hydrogen peroxide from mutant forms of the prion protein fragment PrP121-231.
By means of electron spin resonance spectroscopy, in conjunction with the spin trapping technique, we have shown previously that… 
Highly Cited
2003
Highly Cited
2003
Prion Protein Fragment 106–126 Induces a p38 MAP Kinase—Dependent Apoptosis in SH‐SY5Y Neuroblastoma Cells Independently from the Amyloid Fibril Formation
Abstract: Prion diseases are neurodegenerative disorders of the central nervous system of humans and animals, characterized by… 
2003
2003
Sphingosine Kinase-Dependent Migration of Immature Dendritic Cells in Response to Neurotoxic Prion Protein Fragment
ABSTRACT The concept that circulating dendritic cells mediate neuroinvasion in transmissible spongiform encephalopathies received… 
2001
2001
Channel activity of deamidated isoforms of prion protein fragment 106–126 in planar lipid bilayers
Using the lipid bilayer technique, we have found that age‐related derivatives, PrP[106–126] (L‐Asp108) and PrP[106–126] (L‐iso… 
Highly Cited
2000
Highly Cited
2000
A Nonfibrillar Form of the Fusogenic Prion Protein Fragment [118‐135] Induces Apoptotic Cell Death in Rat Cortical Neurons
Abstract: Neuronal loss is a salient feature of prion diseases.However, its cause and mechanism, particularly its relationship… 
Highly Cited
1998
Highly Cited
1998
Prion protein fragment 106–126 induces apoptotic cell death and impairment of L‐type voltage‐sensitive calcium channel activity in the GH3 cell line
The prion diseases are transmissible neurodegenerative pathologies characterized by the accumulation of altered forms of the… 
Highly Cited
1996
Highly Cited
1996
Intracellular calcium rise through L-type calcium channels, as molecular mechanism for prion protein fragment 106-126-induced astroglial proliferation.
The infectious prion protein (PrPSc) is the etiologic agent of transmissible neurodegenerative conditions such as scrapie or… 
1995
1995
Change in membrane permeability induced by amyloid beta-protein fragment 25-35 in brain neurons dissociated from rats.
Effects of amyloid beta-protein fragment 25-35, A beta P(25-35), on the membrane permeability of organic molecules were examined… 
Highly Cited
1992
Highly Cited
1992
Tau protein induces bundling of microtubules in vitro: Comparison of different tau isoforms and a tau protein fragment
Expression of tau protein in non‐neuronal cells can result in a redistribution of the microtubule cytoskeleton into thick bundles… 
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