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KN 92

Known as: KN-92 
 
National Institutes of Health

Papers overview

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Highly Cited
2011
Highly Cited
2011
Catecholaminergic polymorphic ventricular tachycardia (CPVT) is an inherited arrhythmogenic disease characterized by life… Expand
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Highly Cited
2009
Highly Cited
2009
In the heart, oxidative stress caused by exogenous H2O2 has been shown to induce early afterdepolarizations (EADs) and triggered… Expand
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Highly Cited
2008
Highly Cited
2008
Regulation of genes targeted by the ligand-activated aryl hydrocarbon receptor (AhR) has been shown to be controlled by calcium… Expand
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Highly Cited
2006
Highly Cited
2006
The effect of Ca2+/calmodulin-dependent protein kinase II (CaMK II) on voltage-gated ion channels is widely studied through the… Expand
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2002
2002
KN-93, a Ca2+/calmodulin-dependent protein kinase II (CaMKII) inhibitor, concentration-dependently and reversibly inhibited… Expand
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Highly Cited
2000
Highly Cited
2000
Cardiac beating arises from the spontaneous rhythmic excitation of sinoatrial (SA) node cells. Here we report that SA node… Expand
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Highly Cited
1999
Highly Cited
1999
The present study was undertaken to investigate the effects of specific inhibitors of calmodulin-dependent protein kinase II… Expand
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Highly Cited
1998
Highly Cited
1998
The multifunctional Ca++/calmodulin-dependent protein kinase II (CaM kinase) mediates Ca++-induced augmentation of L-type Ca… Expand
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Highly Cited
1997
Highly Cited
1997
Cultured neonatal ventricular myocytes display features of myocardial hypertrophy including increased cell size, myofilament… Expand
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Highly Cited
1995
Highly Cited
1995
CaMK-II (the type II multifunctional Ca2+/calmodulin kinase) is a ubiquitous serine/threonine protein kinase that is activated by… Expand
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