Skip to search formSkip to main contentSkip to account menu

JEB protocol

Known as: JEB, Bleomycin/Carboplatin/Etoposide, JM8-Etoposide-Bleomycin Regimen 
An abbreviation for a chemotherapy combination used to treat childhood ovarian and testicular germ cell cancers that have spread. It includes the… 
National Institutes of Health

Related topics

Related topics

7 relations

Broader (4)

Antineoplastic Combined Chemotherapy ProtocolsBleomycinCarboplatinEtoposide
Paraplatinpediatric ovarian germ cell tumortesticular germ cell tumor, pediatric

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2006
2006
Sustained phenotypic reversion of junctional epidermolysis bullosa dog keratinocytes: Establishment of an immunocompetent animal model for cutaneous gene therapy.
2006
2006
Evaluation of prenatal intra-amniotic LAMB3 gene delivery in a mouse model of Herlitz disease
Prenatal gene therapy has been considered for Herlitz junctional epidermolysis bullosa (H-JEB), a lethal genodermatosis caused by… 
2005
2005
J.Z. AND THE DISCOVERY OF SQUID GIANT NERVE FIBRES
[⇓][1] ![Figure][2] Richard Keynes writes about J. Z. Young's 1938 ground-breaking publication on the function of squid… 
Highly Cited
2003
Highly Cited
2003
Sustainable correction of junctional epidermolysis bullosa via transposon-mediated nonviral gene transfer
Sustainable correction of severe human genetic disorders of self-renewing tissues, such as the blistering skin disease junctional… 
1998
1998
Cell cycle and adhesion defects in mice carrying a targeted deletion of the integrin beta4 cytoplasmic domain.
The cytoplasmic domain of the integrin beta4 subunit mediates both association with the hemidesmosomal cytoskeleton and… 
1998
1998
Extent of Laminin-5 Assembly and Secretion Effect Junctional Epidermolysis Bullosa Phenotype
Junctional epidermolysis bullosa (JEB) is an autosomal recessive skin blistering disease with both lethal and nonlethal forms… 
1997
1997
Hemidesmosome assembly assessed by expression of a wild-type integrin beta 4 cDNA in junctional epidermolysis bullosa keratinocytes.
The cytoplasmic domain of integrin beta 4, which contains four type III fibronectin-like motifs, seems to be involved in the… 
Highly Cited
1996
Highly Cited
1996
Functional Re-expression of Laminin-5 in Laminin-γ2-deficient Human Keratinocytes Modifies Cell Morphology, Motility, and Adhesion*
Herlitz junctional epidermolysis bullosa (H-JEB) is characterized by a reduced adherence of keratinocytes consequent to deficient… 
1994
1994
The genes for nicein/kalinin 125- and 100-kDa subunits, candidates for junctional epidermolysis bullosa, map to chromosomes 1q32 and 1q25-q31.
Expression of nicein is specifically hampered in the severe form of junctional epidermolysis bullosa (JEB), a recessive… 
1993
1993
Nicein (BM-600) in junctional epidermolysis bullosa: polyclonal antibodies provide new clues for pathogenic role.
We have raised polyclonal antibodies against each of three subunits of the new basement membrane component nicein (formerly BM… 
By clicking accept or continuing to use the site, you agree to the terms outlined in our Privacy Policy (opens in a new tab), Terms of Service (opens in a new tab), and Dataset License (opens in a new tab)