GAD1 gene

Known as: GLUTAMATE DECARBOXYLASE 1, GAD, GAD1 
 
National Institutes of Health

Papers overview

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Highly Cited
2011
Highly Cited
2011
GABA signaling molecules are critical for both human brain development and the pathophysiology of schizophrenia. We examined the… (More)
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Review
2008
Review
2008
Many risk genes interact synergistically to produce schizophrenia and many neurotransmitter interactions have been implicated. We… (More)
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Highly Cited
2008
Highly Cited
2008
BACKGROUND The 65-kD isoform of glutamic acid decarboxylase (GAD) is a major autoantigen in patients with type 1 diabetes… (More)
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Highly Cited
2007
Highly Cited
2007
Cortical GABAergic dysfunction has been implicated as a key component of the pathophysiology of schizophrenia and decreased… (More)
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Highly Cited
2007
Highly Cited
2007
Dysfunction of prefrontal cortex in schizophrenia includes changes in GABAergic mRNAs, including decreased expression of GAD1… (More)
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Review
2006
Review
2006
The 67 and 65 kDa isoforms of glutamic acid decarboxylase, the key enzymes for GABA biosynthesis, are expressed at altered levels… (More)
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Highly Cited
2005
Highly Cited
2005
Postmortem brain studies have shown deficits in the cortical γ-aminobutyric acid (GABA) system in schizophrenic individuals… (More)
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Highly Cited
1999
Highly Cited
1999
The gracile axonal dystrophy (gad) mouse is an autosomal recessive mutant that shows sensory ataxia at an early stage, followed… (More)
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Highly Cited
1997
Highly Cited
1997
Strategies for assessing risk of progression to IDDM, based on single and combined autoantibody measurement, were evaluated in 2… (More)
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Highly Cited
1996
Highly Cited
1996
Islet cell antibodies (ICAs) are predictive of type I diabetes in first-degree relatives, but this immunohistochemical assay has… (More)
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