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CD40 wt Allele
Known as:
Tumor Necrosis Factor Receptor Superfamily, Member 5 Gene
, MGC9013
, CD40 Molecule, TNF Receptor Superfamily Member 5 wt Allele
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Human CD40 wild-type allele is located within 20q12-q13.2 and is approximately 186 kb in length. This allele, which encodes tumor necrosis factor…
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National Institutes of Health
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Related topics
Related topics
15 relations
20q12-q13.2
Apoptosis
Bystander B Cell Activation Pathway
CD40 gene
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Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
2008
2008
Tumor necrosis factor-alpha-mediated suppression of adipocyte apolipoprotein E gene transcription: primary role for the nuclear factor (NF)-kappaB pathway and NFkappaB p50.
L. Yue
,
J. Christman
,
T. Mazzone
Endocrinology
2008
Corpus ID: 42656550
The adipose tissue inflammation accompanying obesity has important consequences for adipocyte lipid metabolism, and increased…
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Highly Cited
2006
Highly Cited
2006
Nickel Compounds Render Anti-apoptotic Effect to Human Bronchial Epithelial Beas-2B Cells by Induction of Cyclooxygenase-2 through an IKKβ/p65-dependent and IKKα- and p50-independent Pathway*
Jin Ding
,
Xinhai Zhang
,
+7 authors
Chuanshu Huang
Journal of Biological Chemistry
2006
Corpus ID: 20025464
The carcinogenicity of nickel compounds has been well documented both in vitro and in vivo; however, the molecular mechanisms by…
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2004
2004
Neuropathological consequences of prenatal cocaine exposure in the mouse
Jia-Qian Ren
,
C. Malanga
,
E. Tabit
,
B. Kosofsky
International Journal of Developmental…
2004
Corpus ID: 25713735
Highly Cited
2002
Highly Cited
2002
NFkappaB1 (p50)-deficient mice are not susceptible to multiple low-dose streptozotocin-induced diabetes.
J. Mabley
,
G. Haskó
,
+4 authors
C. Szabó
Journal of Endocrinology
2002
Corpus ID: 24564348
Insulin-dependent diabetes mellitus (IDDM) is a disease characterized by the autoimmune destruction of the pancreatic beta-cells…
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Highly Cited
1999
Highly Cited
1999
The p65 subunit of NF-kappa B is redundant with p50 during B cell proliferative responses, and is required for germline CH transcription and class switching to IgG3.
Bruce H. Horwitz
,
Bruce H. Horwitz
,
+5 authors
C. Snapper
Journal of Immunology
1999
Corpus ID: 25072472
B cells lacking individual NF-kappa B/Rel family members exhibit defects in activation programs. We generated small resting B…
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Highly Cited
1998
Highly Cited
1998
Transcriptional regulation of intercellular adhesion molecule-1 in astrocytes involves NF–κB and C/EBP isoforms
Sung Joong Lee
,
J. Hou
,
E. Benveniste
Journal of Neuroimmunology
1998
Corpus ID: 19502113
Highly Cited
1996
Highly Cited
1996
Heritability of inhibitory gating of the P50 auditory-evoked potential in monozygotic and dizygotic twins.
David A. Young
,
Merilyne C. Waldo
,
James H. Rutledge
,
Robert Freedman
Neuropsychobiology
1996
Corpus ID: 46786491
Most normal subjects suppress the P50 evoked response to repeated auditory stimulus when paired stimuli are presented, 500 ms…
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Highly Cited
1996
Highly Cited
1996
Differential nuclear localization of p50, p52, and RelB proteins in human accessory cells of the immune response in situ
J. Feuillard
,
M. Körner
,
A. Israel
,
Jeanny Vassy
,
M. Raphael
European Journal of Immunology
1996
Corpus ID: 24677984
The Rel/NF‐ϰB proteins, p50, p52, p65, c‐Rel, and RelB, constitute a family of transcription factors involved in the positive…
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Highly Cited
1993
Highly Cited
1993
Two FKBP-related proteins are associated with progesterone receptor complexes.
D. F. Smith
,
B. Baggenstoss
,
T. Marion
,
R. Rimerman
Journal of Biological Chemistry
1993
Corpus ID: 37730385
Highly Cited
1993
Highly Cited
1993
Retinoic acid induction of major histocompatibility complex class I genes in NTera-2 embryonal carcinoma cells involves induction of NF-kappa B (p50-p65) and retinoic acid receptor beta-retinoid X…
J. Segars
,
T. Nagata
,
+7 authors
T. Tang
Molecular and Cellular Biology
1993
Corpus ID: 26873940
Retinoic acid (RA) treatment of human embryonal carcinoma (EC) NTera-2 (NT2) cells induces expression of major histocompatibility…
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