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BETA-N-ACETYLHEXOSAMINIDASE

Known as: beta N Acetyl hexosaminidase, N-Acetyl-beta-D-hexosaminidase, beta N Acetylhexosaminidases 
A hexosaminidase specific for non-reducing N-acetyl-D-hexosamine residues in N-acetyl-beta-D-hexosaminides. It acts on GLUCOSIDES; GALACTOSIDES; and… Expand
National Institutes of Health

Related topics

Related topics

16 relations
Gangliosidoses, GM2Hexosaminidase A fr SerIn BloodLiver
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Broader (1)

Hexosaminidases

Narrower (1)

peptide O-linked N-acetylglucosamine-beta-N-acetylglucosaminidase

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
Highly Cited
2004
Highly Cited
2004
Lysosomal Glycosphingolipid Recognition by NKT Cells
NKT cells represent a distinct lineage of T cells that coexpress a conserved αβ T cell receptor (TCR) and natural killer (NK… Expand
Highly Cited
2003
Highly Cited
2003
Characterization of novel stem cell factor responsive human mast cell lines LAD 1 and 2 established from a patient with mast cell sarcoma/leukemia; activation following aggregation of FcepsilonRI or…
Two novel stem cell factor (SCF) dependent human mast cell lines, designated LAD 1 and 2, were established from bone marrow… Expand
Highly Cited
2001
Highly Cited
2001
The sortilin cytoplasmic tail conveys Golgi–endosome transport and binds the VHS domain of the GGA2 sorting protein
Sortilin belongs to a growing family of multiligand type‐1 receptors with homology to the yeast receptor Vps10p. Based on… Expand
Highly Cited
2000
Highly Cited
2000
Microglial activation precedes acute neurodegeneration in Sandhoff disease and is suppressed by bone marrow transplantation.
Sandhoff disease is a lysosomal storage disorder characterized by the absence of beta-hexosaminidase and storage of G(M2… Expand
Highly Cited
1997
Highly Cited
1997
Lysosomes Behave as Ca2+-regulated Exocytic Vesicles in Fibroblasts and Epithelial Cells
Lysosomes are considered to be a terminal degradative compartment of the endocytic pathway, into which transport is mostly… Expand
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Highly Cited
1997
Highly Cited
1997
Prevention of lysosomal storage in Tay-Sachs mice treated with N-butyldeoxynojirimycin.
The glycosphingolipid (GSL) lysosomal storage diseases result from the inheritance of defects in the genes encoding the enzymes… Expand
Highly Cited
1984
Highly Cited
1984
Interleukin 3: A differentiation and growth factor for the mouse mast cell that contains chondroitin sulfate E proteoglycan.
Mouse mast cells were differentiated and grown by culturing bone marrow cells in medium containing 2 X 10(-10) M purified… Expand
Highly Cited
1982
Highly Cited
1982
Prostaglandin D2 generation after activation of rat and human mast cells with anti-IgE.
Anti-IgE-dependent activation of rat and human mast cells resulted in the preferential generation of the cyclooxygenase products… Expand
Highly Cited
1981
Highly Cited
1981
Acid hydrolases and tryptase from secretory granules of dispersed human lung mast cells.
beta-Hexosaminidase, beta-glucuronidase, arylsulfatase, and tryptase were each released along with histamine from dispersed… Expand
Highly Cited
1969
Highly Cited
1969
Tay-Sachs Disease: Generalized Absence of a Beta-D-N-Acetylhexosaminidase Component
Two hexosaminidase components, separable by starch-gel electrophoresis and possessing both β-D-N-acetylglucosaminidase and β-D-N… Expand
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