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UCP 0054287

Known as: UCP-0054287, UCP0054287 
 
National Institutes of Health

Related topics

Related topics

2 relations

Broader (2)

BenzamidesBenzodioxoles

Papers overview

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2017
2017
Field-friendly serological tests for determination of M. leprae-specific antibodies
Early detection of leprosy is key to reduce the ongoing transmission. Antibodies directed against M. leprae PGL-I represent a… Expand
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2015
2015
Antioxidant treatment normalizes mitochondrial energetics and myocardial insulin sensitivity independently of changes in systemic metabolic homeostasis in a mouse model of the metabolic syndrome.
Cardiac dysfunction in obesity is associated with mitochondrial dysfunction, oxidative stress and altered insulin sensitivity… Expand
2015
2015
Metabolizable Protein and Energy Requirements for Lactating Buffaloes fed on Silage Based Diets.
The present study was conducted to determine the metabolizable energy (ME) and protein (MP) requirements of lactating buffaloes… Expand
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2013
2013
TRPV1-mediated UCP2 upregulation ameliorates hyperglycemia-induced endothelial dysfunction
BackgroundDiabetic cardiovascular complications are characterised by oxidative stress-induced endothelial dysfunction. Uncoupling… Expand
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2003
2003
Self-selected macronutrient diet affects energy and glucose metabolism in brown fat-ablated mice.
OBJECTIVE Obese transgenic UCP-DTA mice have largely ablated brown adipose tissue and develop obesity and diabetes, which are… Expand
Highly Cited
1998
Highly Cited
1998
Severe Leptin Resistance in Brown Fat-Deficient Uncoupling Protein Promoter-Driven Diphtheria Toxin A Mice Despite Suppression of Hypothalamic Neuropeptide Y and Circulating Corticosterone…
Brown adipose tissue (BAT) has the capacity for uncoupled mitochondrial respiration and is proposed to be a key site for… Expand
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Highly Cited
1998
Highly Cited
1998
Role of neuropeptide Y in diet-, chemical- and genetic-induced obesity of mice
OBJECTIVE: The goal of this study was to ascertain whether neuropeptide Y (NPY) is required in mice for the development of… Expand
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1998
1998
Temperature-dependent feeding: lack of role for leptin and defect in brown adipose tissue-ablated obese mice.
The objective was to characterize the ability of control and transgenic brown adipose tissue (BAT)-ablated uncoupling protein… Expand
1998
1998
Temperature-dependent feeding: lack of role for leptin and defect in brown adipose tissue-ablated obese mice.
The objective was to characterize the ability of control and transgenic brown adipose tissue (BAT)-ablated uncoupling protein… Expand
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Highly Cited
1997
Highly Cited
1997
Raising at thermoneutrality prevents obesity and hyperphagia in BAT-ablated transgenic mice.
Transgenic mice with ablation of brown adipocytes induced by brown adipocyte-specific expression of diphtheria toxin A chain (DTA… Expand
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