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Tiapamil

National Institutes of Health

Papers overview

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Highly Cited
1991
Highly Cited
1991
Resistance of human cancer cells to multiple cytotoxic hydrophobic agents (multidrug resistance) is due to overexpression of the… Expand
Review
1988
Review
1988
SummaryCalcium antagonist drugs under clinical development are of the Type I (verapamil, diltiazem-like) and Type II (nifedipine… Expand
Highly Cited
1988
Highly Cited
1988
A marked increase in cellular ATP consumption was induced by verapamil in the multidrug-resistant (MDR) cell line 2780AD, but not… Expand
Review
1987
Review
1987
The Ca2+ channel blockers, diltiazem, nifedipine, and vcrapamil, are now well-established members of the therapeutic… Expand
1986
1986
Summary: To clarify the pharmacological profile of the two new calcium channel blockers tiapamil and nisoldipine in humans, their… Expand
1985
1985
Studies were performed with the anthracycline-responsive P388 murine leukemia cell line, and with P388/ADR, a subline selected… Expand
Highly Cited
1983
Highly Cited
1983
[3H]Nitrendipine binding to drug receptor sites associated with calcium channels is allosterically regulated by a diverse group… Expand
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1983
1983
The investigation was designed to test whether nicardipine and tiapamil interact with digoxin, resulting in increased plasma… Expand
Highly Cited
1983
Highly Cited
1983
The binding of the Ca2+‐channel blocker d‐cis‐[3H]diltiazem to guinea pig skeletal muscle microsomes is temperature‐dependent. At… Expand
1982
1982
  • G. Wendt
  • Cardiology
  • 1982
  • Corpus ID: 19533633
The main pathways of biotransformation of tiapamil and basic pharmacokinetic information on the drug are presented and compared… Expand