SEMA3F gene

Known as: SEMAK, SEMAPHORIN III/F, sema IV 
This gene is involved in both cell signaling and axon guidance.
National Institutes of Health

Topic mentions per year

Topic mentions per year

1996-2016
051019962016

Papers overview

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2012
2012
Inactivation of tumor suppressors and inhibitory microenvironmental factors is necessary for breast cancer invasion; therefore… (More)
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2008
2008
Class three semaphorins (SEMAs) were originally shown to be mediators of axon guidance that repelled axons and collapsed growth… (More)
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2007
2007
Loss of SEMA3F occurs frequently in lung cancer and correlates with advanced stage of disease. We previously reported that SEMA3F… (More)
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Highly Cited
2004
Highly Cited
2004
Melanoma is the most lethal skin cancer. Most deaths from melanoma result from metastases. Semaphorins have been shown to inhibit… (More)
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2003
2003
SEMA3F, isolated from a 3p21.3 deletion, has antitumor activity in transfected cells, and protein expression correlates with… (More)
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Highly Cited
2003
Highly Cited
2003
Two receptors, neuropilin 1 (NP1) and neuropilin 2 (NP2), bind class 3 semaphorins, axon guidance molecules including SEMA3F, the… (More)
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Highly Cited
2002
Highly Cited
2002
Loss of heterozygosity on human chromosome 3p21.3 is a frequent occurrence in many tumor types. In a previous study, our… (More)
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2000
2000
Semaphorins/collapsins are a family of secreted and membrane-associated proteins involved in nerve growth cone migration. However… (More)
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1999
1999
During development, growth cones can be guided at a distance by diffusible factors, which are attractants and/or repellents. The… (More)
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1996
1996
Small cell lung cancer (SCLC) has been correlated with a deletion in the short arm of chromosome 3, with the region 3p21 being… (More)
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