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SC 560

Known as: SC-560, SC560 
 
National Institutes of Health

Papers overview

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Highly Cited
2008
Highly Cited
2008
BackgroundCOX inhibitors and β-blockers were recently suggested to reduce cancer progression through inhibition of tumor… Expand
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Highly Cited
2006
Highly Cited
2006
Local increase in blood flow during neural activity forms the basis for functional brain imaging, but its mechanism remains… Expand
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Highly Cited
2005
Highly Cited
2005
Using intracerebral microdialysis, we reported previously that acute in vivo activation of NMDA glutamate receptors triggers… Expand
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Highly Cited
2002
Highly Cited
2002
COX2-selective non-steroidal anti-inflammatory drugs (NSAIDs) cause selective apoptosis of renal medullary interstitial cells… Expand
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Highly Cited
2002
Highly Cited
2002
A recent study demonstrated that inhibition of both cyclooxygenase (COX)‐1 and COX‐2 is required for the development of… Expand
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Highly Cited
2001
Highly Cited
2001
The regular use of various nonsteroidal anti‐inflammatory drugs (NSAIDs) was shown to decrease the incidence of colorectal cancer… Expand
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Highly Cited
2001
Highly Cited
2001
The clinical use of doxorubicin, an anthracycline chemotherapeutic agent, is limited by cardiotoxicity, particularly when… Expand
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Highly Cited
2000
Highly Cited
2000
BACKGROUND & AIMS Selective cyclooxygenase (COX)-2 inhibitors produce less gastric damage than conventional nonsteroidal anti… Expand
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Highly Cited
1998
Highly Cited
1998
The enzymes cyclooxygenase-1 and cyclooxygenase-2 (COX-1 and COX-2) catalyze the conversion of arachidonic acid to prostaglandin… Expand
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Highly Cited
1991
Highly Cited
1991
Genetic and molecular data now available on the pathogenic properties of Shigella flexneri allow rational design of live… Expand
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