RIPK3 gene

Known as: RIPK3, RIP3, RECEPTOR-INTERACTING PROTEIN 3 
This gene plays a role in signal transduction and the activation of apoptosis.
National Institutes of Health

Papers overview

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Review
2017
Review
2017
Necroptosis is a form of regulated cell death, which is induced by ligand binding to TNF family death domain receptors, pattern… (More)
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2017
2017
Ripk3-required necroptosis and mitochondria-mediated apoptosis are the predominant types of cell death that largely account for… (More)
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Highly Cited
2016
Highly Cited
2016
Necroptosis is a caspase-independent form of cell death that is triggered by activation of the receptor interacting serine… (More)
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Highly Cited
2015
Highly Cited
2015
RIPK3 and its substrate MLKL are essential for necroptosis, a lytic cell death proposed to cause inflammation via the release of… (More)
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Highly Cited
2014
Highly Cited
2014
Receptor-interacting protein kinase (RIPK)-1 is involved in RIPK3-dependent and -independent signaling pathways leading to cell… (More)
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Highly Cited
2014
Highly Cited
2014
Receptor-interacting protein kinase 1 (RIPK1) and RIPK3 trigger pro-inflammatory cell death termed “necroptosis.” Studies with… (More)
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Highly Cited
2014
Highly Cited
2014
Necroptosis is a form of programmed cell death that depends on the activation of receptor interacting protein kinase-1 (RIPK1… (More)
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Highly Cited
2013
Highly Cited
2013
Receptor-interacting protein kinase (RIPK) 1 and RIPK3 have emerged as essential kinases mediating a regulated form of necrosis… (More)
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Highly Cited
2013
Highly Cited
2013
Ligation of tumor necrosis factor receptor 1 (TNFR1) can cause cell death by caspase 8 or receptor-interacting protein kinase 1… (More)
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Highly Cited
2009
Highly Cited
2009
Necrosis can be induced by stimulating death receptors with tumor necrosis factor (TNF) or other agonists; however, the… (More)
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