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MK-329

Known as: MK 329, MK329 
National Institutes of Health

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1 relation

Broader (1)

Devazepide

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
1994
1994
Role of cholecystokinin in the motivational control of instrumental action in rats.
In 4 experiments, the role of cholecystokinin (CCK) in the motivational control of instrumental performance in rats was assessed… 
1993
1993
Effect of cholecystokinin and secretin on somatostatin release from cultured antral cells.
1993
1993
Cholecystokinin increases intracellular Ca2+ concentration in the Human JURKAT T Lymphocyte cell line.
Highly Cited
1992
Highly Cited
1992
A Pilot Clinical Trial of the Cholecystokinin Receptor Antagonist MK‐329 in Patients with Advanced Pancreatic Cancer
MK-329 is a nonpeptidal, highly specific cholecystokinin (CCK) receptor antagonist, with affinity for pancreatic and gallbladder… 
1992
1992
Mediation of trypsin inhibitor-induced pancreatic hypersecretion by secretin and cholecystokinin in rats.
Highly Cited
1991
Highly Cited
1991
Type-A CCK receptors mediate the inhibition of food intake and activity by CCK-8 in 9- to 12-day-old rat pups
1991
1991
Inhibition of cholecystokinin-stimulated pancreaticobiliary output in man by the cholecystokinin receptor antagonist MK-329.
MK-329 (formerly L-364,718) is a new nonpeptide antagonist for the peripheral (type-A) cholecystokinin (CCK) receptor, which has… 
Highly Cited
1990
Highly Cited
1990
Reversal of the anorectic effect of (+)‐fenfluramine in the rat by the selective cholecystokinin receptor antagonist MK‐329
1 Experiments were conducted to determine whether or not the effect of (+)‐fenfluramine (3.0 mg kg−1, i.p.) on food intake can be… 
1990
1990
Regulation of pancreatic endocrine function by cholecystokinin: studies with MK-329, a nonpeptide cholecystokinin receptor antagonist.
A cholecystokinin (CCK) receptor antagonist, MK-329, was used to explore the physiological role of CCK in regulating pancreatic… 
Highly Cited
1990
Highly Cited
1990
Endogenous cholecystokinin reduces feeding in young rats.
The hypothesis that endogenous cholecystokinin (CCK) released from the small intestine during feeding causes satiety was tested… 
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