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L 158809

Known as: L-158,809, L-158809 
National Institutes of Health

Papers overview

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Highly Cited
2003
Highly Cited
2003
Abstract—Enhanced tissue angiotensin (Ang) II levels have been reported in diabetes and might lead to cardiac dysfunction through… Expand
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Highly Cited
2002
Highly Cited
2002
The current therapy for chronic proteinuric nephropathies is angiotensin-converting enzyme inhibitors (ACEi), which slow, but may… Expand
Highly Cited
2001
Highly Cited
2001
Angiotensin-converting enzyme inhibitors restore size-selective dysfunction of the glomerular barrier in experimental animals and… Expand
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Highly Cited
2000
Highly Cited
2000
PURPOSE This report summarizes our experiences on the protective effect of angiotensin-converting enzyme (ACE) inhibitors… Expand
Highly Cited
1999
Highly Cited
1999
Angiotensin II type 1 (AT(1)) receptors are located on pressor neurons in the rostral ventrolateral medulla, and their activation… Expand
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Highly Cited
1997
Highly Cited
1997
Enhanced sympathetic outflow is seen in both patients with congestive heart failure and animals with experimental heart failure… Expand
Highly Cited
1996
Highly Cited
1996
Although angiotensin II (Ang II) and the heptapeptide Ang-(1-7) differ by only one amino acid, the two peptides produce different… Expand
Highly Cited
1996
Highly Cited
1996
To gain insight into the contribution of immunologic and hemodynamic factors in the progressive demise of structure and function… Expand
Review
1992
Review
1992
Angiotensin II (ANG II) is the primary mediator of the renin-angiotensin system, which has an important functional role in… Expand
Highly Cited
1992
Highly Cited
1992
Previous studies in experimental models of progressive renal failure have shown that the capacity of antihypertensive drugs to… Expand