Glomerulonephritis, Membranoproliferative

Known as: Membranoproliferative glomerulonephritis NOS, Mesangiocapillary Glomerulonephritides, Mesangiocapillary Glomerulonephritis 
Chronic glomerulonephritis characterized histologically by proliferation of MESANGIAL CELLS, increase in the MESANGIAL EXTRACELLULAR MATRIX, and a… (More)
National Institutes of Health

Topic mentions per year

Topic mentions per year

1951-2018
05010019512017

Related topics

Related topics

16 relations
Chronic inflammationGlomerulonephritisIn BloodKidney
(More)

Narrower (4)

Glomerulonephritis sparse hair telangiectasesGlomerulopathy with fibronectin depositsMembranoproliferative Glomerulonephritis, Type IMembranoproliferative Glomerulonephritis, Type III

Related mentions per year

Related mentions per year

1936-2019
19401960198020002020
Glomerulonephritis, Membranoproliferative
Kidney
Microbiological
Glomerulonephritis
prevention & control
Chronic inflammation

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
Highly Cited
2012
Highly Cited
2012
Eculizumab and refractory membranoproliferative glomerulonephritis.
n engl j med 366;12 nejm.org march 22, 2012 1165 sions associated with dense-deposit disease.1 Our observations show that at… (More)
  • figure 1
Is this relevant?
Highly Cited
2011
Highly Cited
2011
Proliferative glomerulonephritis secondary to dysfunction of the alternative pathway of complement.
BACKGROUND AND OBJECTIVES dense deposit disease (DDD) is the prototypical membranoproliferative glomerulonephritis (MPGN), in… (More)
  • table 1
  • figure 1
  • figure 2
  • figure 3
  • table 4
Is this relevant?
Highly Cited
2007
Highly Cited
2007
Primary glomerulonephritis with isolated C3 deposits: a new entity which shares common genetic risk factors with haemolytic uraemic syndrome.
INTRODUCTION Abnormal control of the complement alternative pathway (CAP) (factor H, factor I and membrane cofactor protein (MCP… (More)
  • figure 1
  • figure 2
  • table 1
  • table 2
  • table 3
Is this relevant?
Highly Cited
2006
Highly Cited
2006
Variations in the complement regulatory genes factor H (CFH) and factor H related 5 (CFHR5) are associated with membranoproliferative glomerulonephritis type II (dense deposit disease).
INTRODUCTION Membranoproliferative glomerulonephritis type II or dense deposit disease (MPGN II/DDD) causes chronic renal… (More)
Is this relevant?
Highly Cited
2004
Highly Cited
2004
Heterozygous and homozygous factor h deficiencies associated with hemolytic uremic syndrome or membranoproliferative glomerulonephritis: report and genetic analysis of 16 cases.
Factor H (FH) is the major regulatory protein of the complement alternative pathway, with a structure consisting of a tandem… (More)
  • table 1
  • table 2
  • table 3
  • figure 1
  • table 4
Is this relevant?
Highly Cited
2001
Highly Cited
2001
Structure and composition of drusen associated with glomerulonephritis: Implications for the role of complement activation in drusen biogenesis
Purpose The ocular fundi of many patients with membranoproliferative glomerulonephritis type II (MPGN-II) are characterised by… (More)
  • figure 1
  • table 1
  • figure 2
Is this relevant?
Highly Cited
2000
Highly Cited
2000
Functional loss of ABCA1 in mice causes severe placental malformation, aberrant lipid distribution, and kidney glomerulonephritis as well as high-density lipoprotein cholesterol deficiency.
Tangier disease (TD) and familial HDL deficiency (FHA) have recently been linked to mutations in the human ATP-binding cassette… (More)
Is this relevant?
Highly Cited
1997
Highly Cited
1997
IgG subclasses in patients with membranoproliferative glomerulonephritis, membranous nephropathy, and lupus nephritis.
Primary glomerulopathy can be classified into seven essential patterns based on histopathological studies. The pathogenesis of… (More)
Is this relevant?
Highly Cited
1995
Highly Cited
1995
Hereditary porcine membranoproliferative glomerulonephritis type II is caused by factor H deficiency.
We have recently described hereditary membranoproliferative glomerulonephritis type II in the pig. All affected animals had… (More)
  • figure 1
  • figure 3
  • figure 2
  • figure 4
  • figure 5
Is this relevant?
Highly Cited
1990
Highly Cited
1990
Anti-neutrophil cytoplasmic autoantibodies induce neutrophils to degranulate and produce oxygen radicals in vitro.
Anti-neutrophil cytoplasmic autoantibodies (ANCA) are in the circulation of most patients with pauci-immune necrotizing… (More)
Is this relevant?