Epidermal hyperplasia

 
National Institutes of Health

Papers overview

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Highly Cited
2011
Highly Cited
2011
Tumors and associated stroma manifest mechanical properties that promote cancer. Mechanosensation of tissue stiffness activates… (More)
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Highly Cited
2011
Highly Cited
2011
IL-23 and Th17 cells producing IL-17A and IL-22 are found in excess in skin affected by psoriasis. Previous studies showed that… (More)
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Highly Cited
2009
Highly Cited
2009
T cells are crucial mediators of the skin damage in psoriasis. We here show that interleukin-21 (IL-21), a T cell–derived… (More)
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Highly Cited
2008
Highly Cited
2008
A broad range of experimental and clinical evidence has highlighted the central role of chronic inflammation in promoting tumor… (More)
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Highly Cited
2007
Highly Cited
2007
Biological agents have dramatically improved treatment options for patients with severe psoriasis. Etanercept (tumor necrosis… (More)
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2007
2007
Various common signaling pathways maintain tissue stem cells, including Notch and Wnt/β-catenin signals. Phosphoinositide-3… (More)
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Highly Cited
2006
Highly Cited
2006
Aberrant cytokine expression has been proposed as an underlying cause of psoriasis, although it is unclear which cytokines play… (More)
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Highly Cited
2003
Highly Cited
2003
PTEN is a tumor suppressor gene mutated in many human cancers. We used the Cre-loxP system to generate a keratinocyte-specific… (More)
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Highly Cited
2003
Highly Cited
2003
Caveolin-1 is the principal protein component of caveolae membrane domains, which are located at the cell surface in most cell… (More)
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Highly Cited
1993
Highly Cited
1993
Epithelial-mesenchymal interactions control epidermal growth and differentiation, but little is known about the mechanisms of… (More)
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