Crescentic glomerulonephritis

Known as: GLOMERULONEPHRITIS, EXTRACAPILLARY, Idiopathic Crescentic Glomerulonephritis 
A histopathologic term for a pattern of diseases characterized by extensive crescent formation in the glomeruli; patients present clinically with… (More)
National Institutes of Health

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KidneyKidney GlomerulusRapidly Progressive GlomerulonephritisUrinary system

Papers overview

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Highly Cited
2009
Highly Cited
2009
C5a receptor mediates neutrophil activation and ANCA-induced glomerulonephritis.
Anti-neutrophil cytoplasmic autoantibody (ANCA)-induced necrotizing crescentic glomerulonephritis (NCGN) requires complement… (More)
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Highly Cited
2006
Highly Cited
2006
T cells in crescentic glomerulonephritis.
Crescent formation in glomerulonephritis (GN) is a manifestation of severe glomerular injury that usually results in a poor… (More)
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Highly Cited
2005
Highly Cited
2005
Conditional ablation of macrophages halts progression of crescentic glomerulonephritis.
The presence of macrophages in inflamed glomeruli of rat kidney correlates with proliferation and apoptosis of resident… (More)
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Highly Cited
2004
Highly Cited
2004
Immune complex deposits in ANCA-associated crescentic glomerulonephritis: a study of 126 cases.
BACKGROUND Necrotizing and crescentic glomerulonephritis related to antineutrophil cytoplasmic autoantibodies (ANCA) is typically… (More)
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Highly Cited
2004
Highly Cited
2004
Podocytes populate cellular crescents in a murine model of inflammatory glomerulonephritis.
Cellular crescents are a defining histologic finding in many forms of inflammatory glomerulonephritis. Despite numerous studies… (More)
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Highly Cited
2002
Highly Cited
2002
Antineutrophil cytoplasmic autoantibodies specific for myeloperoxidase cause glomerulonephritis and vasculitis in mice.
Antineutrophil cytoplasmic autoantibodies (ANCAs) are identified in the circulation of approximately 80% of patients with pauci… (More)
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Highly Cited
2002
Highly Cited
2002
WT1 is a key regulator of podocyte function: reduced expression levels cause crescentic glomerulonephritis and mesangial sclerosis.
Glomerular disease is one of the most common causes of end-stage renal failure. Increasing evidence suggests that these… (More)
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Highly Cited
2000
Highly Cited
2000
Protease-Activated Receptor 1 Mediates Thrombin-Dependent, Cell-Mediated Renal Inflammation in Crescentic Glomerulonephritis
Protease-activated receptor (PAR)-1 is a cellular receptor for thrombin that is activated after proteolytic cleavage. The… (More)
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Highly Cited
1997
Highly Cited
1997
Th1 responsiveness to nephritogenic antigens determines susceptibility to crescentic glomerulonephritis in mice.
The pattern of glomerulonephritis (GN) developing in response to a planted antigen (sheep anti-mouse GBM globulin) was compared… (More)
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Highly Cited
1996
Highly Cited
1996
Intervention of crescentic glomerulonephritis by antibodies to monocyte chemotactic and activating factor (MCAF/MCP-1).
We investigated the pathophysiological role of a potent macrophage (M(phi)) chemotactic cytokine (chemokine), monocyte… (More)
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