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Complement C3a measurement

Known as: ASP, Complement C3 DesArg, Acetylation-stimulating Protein, C3A, Complement C3a 
The determination of the amount of complement C3a present in a sample.
National Institutes of Health

Related topics

Related topics

4 relations
C3 geneCDISC SDTM Laboratory Test Terminology by CodeCDISC SDTM Laboratory Test Terminology by NameClinical Data Interchange Standards Consortium Terminology

Papers overview

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2016
2016
Complement C3a Mobilizes Dental Pulp Stem Cells and Specifically Guides Pulp Fibroblast Recruitment.
INTRODUCTION Complement activation is considered a major mechanism in innate immunity. Although it is mainly involved in… Expand
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2015
2015
Knock out CD44 in reprogrammed liver cancer cell C3A increases CSCs stemness and promotes differentiation
CD44 is a widely known cancer stem cells marker in various cancers and validated to function in tumor growth, survival and tumor… Expand
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2013
2013
Matrix stiffness in three-dimensional systems effects on the behavior of C3A cells.
The purpose of this study was to evaluate in vitro how the modulation of stiffness in a three-dimensional (3D) system… Expand
2010
2010
Identification of complement C3f‐desArg and its derivative for acute leukemia diagnosis and minimal residual disease assessment
Therapeutic conditions for acute leukemia (AL) mainly rely on diagnosis and detection of minimal residual disease (MRD). However… Expand
Highly Cited
2009
Highly Cited
2009
DEFECTIVE ENGRAFTMENT OF C3aR-/- HEMATOPOIETIC STEM PROGENITOR CELLS REVEALS A NOVEL ROLE OF THE C3a-C3aR AXIS IN BONE MARROW HOMING
We reported that complement (C) becomes activated and cleaved in bone marrow during preconditioning for hematopoietic… Expand
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2009
2009
Complement Split Products C3a and C4a in Chronic Lyme Disease
Complement split products C3a and C4a are reportedly elevated in patients with acute Lyme disease. We have now examined these… Expand
2008
2008
Complement Split Products C 3 a and C 4 a in Chronic Lyme Disease
Lyme disease caused by the spirochete Borrelia burgdorferi is the most common tick-borne illness in the world today [1, 2… Expand
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Highly Cited
2005
Highly Cited
2005
Complement C3a Enhances CXCL12 (SDF-1)-Mediated Chemotaxis of Bone Marrow Hematopoietic Cells Independently of C3a Receptor1
Complement C3a promotes CXCL12-induced migration and engraftment of human and murine hemopoietic progenitor cells, suggesting a… Expand
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2005
2005
C5L2 receptor is not involved in C3a / C3a-desArg-mediated enhancement of bone marrow hematopoietic cell migration to CXCL12
C5L2 receptor is not involved in C3a / C3a-desArg-mediated enhancement of bone marrow hematopoietic cell migration to CXCL12 
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Highly Cited
1997
Highly Cited
1997
Regulation of B cell functions by C3a and C3a(desArg): suppression of TNF-alpha, IL-6, and the polyclonal immune response.
Regulation of the humoral immune response by fragments of complement component C3 has been shown to play an important role in… Expand
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