CV-11974

Known as: CV 11974, CV11974 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

1993-2017
05101519932017

Papers overview

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Highly Cited
2000
Highly Cited
2000
BACKGROUND AND PURPOSE Angiotensin II, through stimulation of AT(1) receptors, not only controls blood pressure but also… (More)
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Highly Cited
2000
Highly Cited
2000
BACKGROUND We demonstrated earlier that angiotensin II (Ang II), by AT(1) receptor activation, upregulates oxidized LDL (ox-LDL… (More)
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Highly Cited
1999
Highly Cited
1999
This study was designed to determine whether mechanical stretch activates the Janus kinase (JAK)/signal transducers and… (More)
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Highly Cited
1999
Highly Cited
1999
Recent studies suggest that atherosclerosis is a kind of inflammatory process and that cytokine plays important roles in this… (More)
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1998
1998
This study was designed to demonstrate the characteristic pattern of angiotensin II-induced JAK/STAT (indicating just another… (More)
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1997
1997
The mechanisms of the insurmountable antagonism of 2-ethoxy-1-[[2'-(1H-tetrazol-5-yl)biphenyl-4-yl]methyl]1H-benzimid azole -7… (More)
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Highly Cited
1997
Highly Cited
1997
Although angiotensin II type 2 (AT2) receptor has recently been cloned, its functional role is not well understood. We tested the… (More)
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Highly Cited
1996
Highly Cited
1996
Angiotensin-(1-7) [Ang-(1-7)] was recently recognized to have novel biological functions that are distinct from those of Ang II… (More)
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1995
1995
OBJECTIVE To investigate whether the angiotensin II (Ang II) subtype 1 receptor (AT1) antagonist CV-11974 had a similar effect to… (More)
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1993
1993
The angiotensin II (AII) antagonistic action of CV-11974 (2-ethoxy-1-[[2'-(1H-tetrazol-5-yl)biphenyl-4-yl]methyl] benzimidazole-7… (More)
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