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CCAAT/Enhancer Binding Protein Alpha

Known as: C/EBP Alpha, C-EBP alpha, C EBPalpha 
A CCAAT-enhancer-binding protein found in LIVER; ADIPOSE TISSUE; INTESTINES; LUNG; ADRENAL GLANDS; PLACENTA; OVARY and peripheral blood mononuclear… 
National Institutes of Health

Related topics

Related topics

24 relations
Acute Myeloid Leukemia with Mutated CEBPAAndrogen Receptor Regulation PathwayCCAAT-Enhancer-Binding ProteinsCEBPA gene

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
Review
2020
Review
2020
CCAAT enhancer binding protein alpha (CEBPA) biallelic acute myeloid leukaemia: cooperating lesions, molecular mechanisms and clinical relevance
Recent advances in sequencing technologies have allowed for the identification of recurrent mutations in acute myeloid leukaemia… 
Highly Cited
2016
Highly Cited
2016
In Vivo toxicological assessment of biologically synthesized silver nanoparticles in adult Zebrafish (Danio rerio).
Highly Cited
2013
Highly Cited
2013
CG methylated microarrays identify a novel methylated sequence bound by the CEBPB|ATF4 heterodimer that is active in vivo
To evaluate the effect of CG methylation on DNA binding of sequence-specific B-ZIP transcription factors (TFs) in a high… 
Highly Cited
2010
Highly Cited
2010
Complexity of miR-223 regulation by CEBPA in human AML.
Highly Cited
2010
Highly Cited
2010
MicroRNA expression in human retinal pigment epithelial (ARPE-19) cells: Increased expression of microRNA-9 by N-(4-Hydroxyphenyl)retinamide
Purpose MicroRNAs (miRNAs) are important regulators of many cellular functions due to their ability to target mRNAs for… 
Highly Cited
2010
Highly Cited
2010
Inference of hierarchical regulatory network of estrogen-dependent breast cancer through ChIP-based data
BackgroundGlobal profiling of in vivo protein-DNA interactions using ChIP-based technologies has evolved rapidly in recent years… 
2006
2006
Involvement of Endoplasmic Reticulum Stress in Hereditary Tyrosinemia Type I*
Hereditary tyrosinemia type I (HTI) is the most severe disease of the tyrosine degradation pathway. HTI is caused by a deficiency… 
Highly Cited
2004
Highly Cited
2004
The leukemic fusion gene AML1-MDS1-EVI1 suppresses CEBPA in acute myeloid leukemia by activation of Calreticulin.
The leukemic fusion gene AML1-MDS1-EVI1 (AME) encodes a chimeric transcription factor that results from the t(3,21)(q26;q22… 
Highly Cited
2001
Highly Cited
2001
Synergism between ERalpha transactivation function 1 (AF-1) and AF-2 mediated by steroid receptor coactivator protein-1: requirement for the AF-1 alpha-helical core and for a direct interaction…
The transcriptional activity of ERalpha (or NR3A1) after binding of ligand is mediated through synergistic action between… 
Highly Cited
2001
Highly Cited
2001
Genomic characterization, localization, and functional expression of FGL2, the human gene encoding fibroleukin: a novel human procoagulant.
For diseases in which thrombosis plays a pivotal role, such as virus-induced fulminant hepatitis, fetal loss syndrome, and… 
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