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Bleomycin/Prednisone/Vincristine regimen (BOP)

Known as: BLEO/PRED/VCR, BOP, Bleomycin/Prednisone/Vincristine 
National Institutes of Health

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3 relations
BleomycinPrednisoneVincristine

Papers overview

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Review
2012
Review
2012
Adjunctive daily supplementation with encapsulated fruit, vegetable and berry juice powder concentrates and clinical periodontal outcomes: a double-blind RCT
Aim A double-blind randomized controlled trial to determine whether dietary supplementation with fruit/vegetable/berry juice… 
Highly Cited
2011
Highly Cited
2011
Subgingival debridement of periodontal pockets by air polishing in comparison with ultrasonic instrumentation during maintenance therapy.
AIM The objective was to determine clinical and microbiological effects and perceived treatment discomfort of root debridement by… 
Highly Cited
2002
Highly Cited
2002
Safety and clinical effects of topical histatin gels in humans with experimental gingivitis.
BACKGROUND Our research group has recently reported that exogenously applied histatins can inhibit plaque accumulation and… 
Highly Cited
1998
Highly Cited
1998
Intensive induction-sequential chemotherapy with BOP/VIP-B compared with treatment with BEP/EP for poor-prognosis metastatic nonseminomatous germ cell tumor: a Randomized Medical Research Council…
PURPOSE The aim of this randomized trial was to assess the potential therapeutic advantage of an intensive induction-sequential… 
Highly Cited
1996
Highly Cited
1996
SHORT COMMUNICATION: Chemopreventive effects of phenethy1 isothiocyanate on lung and pancreatic tumorigenesis inN-nitrosobis(2-oxopropyl)amine-treated hamsters
The chemopreventive effects of phenethyl isothiocyanate (PEITC) were investigated in N-nitrosobis(2-oxopropyl)-amine (BOP… 
Highly Cited
1993
Highly Cited
1993
K‐ras Gene Mutation in Early Ductal Lesions Induced in a Rapid Production Model for Pancreatic Carcinomas in Syrian Hamsters
The presence of K‐ras gene mutation was examined in experimentally induced preneoplastic pancreatic ductal lesions. Syrian… 
Highly Cited
1989
Highly Cited
1989
Programmed cell death (apoptosis) in pancreatic cancers of hamsters after treatment with analogs of both luteinizing hormone-releasing hormone and somatostatin.
Female Syrian golden hamsters with N-nitrosobis(2-oxopropyl)amine (BOP)-induced ductal pancreatic cancers were treated with long… 
Highly Cited
1989
Highly Cited
1989
Membrane Receptors for Peptides in Experimental and Human Pancreatic Cancers
Membrane receptors for [D-Trp6]-luteinizing hormone-releasing hormone ([D-Trp6-LH-RH), somatostatin (SS-14), and epidermal growth… 
Highly Cited
1985
Highly Cited
1985
Pancreatic carcinogenesis-enhancement by cholecystokinin in the hamster-nitrosamine model.
The role of the pancreaticotrophic hormone cholecystokinin (CCK) in modifying the pancreatic response to carcinogen has been… 
Highly Cited
1978
Highly Cited
1978
Selective induction of pancreatic ductular tumors by single doses of N-nitrosobis(2-oxopropyl)amine in Syrian golden hamsters.
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