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Antimycin A1

 
National Institutes of Health

Papers overview

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Highly Cited
2014
Highly Cited
2014
Mitochondrial dysfunction is a common feature in neurodegeneration and aging. We identify mitochondrial dysfunction in xeroderma… Expand
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2011
2011
Different cell culture models were already used to analyze the molecular base of the neuroprotective activities of the Ginkgo… Expand
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2006
2006
We identified antimycin A1 as an inhibitor of the hypoxia-response element (HRE) from screening using a reporter under the… Expand
Highly Cited
2003
Highly Cited
2003
We investigated how inhibition of mitochondrial Ca2+ uptake affects transmitter release from mouse motor terminals during brief… Expand
Highly Cited
2000
Highly Cited
2000
Increases in cytosolic [Ca2+] evoked by trains of action potentials (20–100 Hz) were recorded from mouse and lizard motor nerve… Expand
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Highly Cited
1997
Highly Cited
1997
Mitochondria act as potent buffers of intracellular Ca2+ in many cells, but a more active role in modulating the generation of… Expand
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Highly Cited
1996
Highly Cited
1996
1. Buffering of glutamate-induced Ca2+ loads in single rat hippocampal neurons grown in primary culture was studied with… Expand
Highly Cited
1995
Highly Cited
1995
The exposure of cultured cerebellar granule cells for 4 min to glutamate (50 microM) in a Mg2+-free medium containing 10 microM… Expand
Highly Cited
1989
Highly Cited
1989
Chemicals with antimalarial effects show different patterns against ring stages, trophozoites, and schizonts of Plasmodium… Expand
1969
1969
  • A. Hussain
  • Journal of pharmaceutical sciences
  • 1969
  • Corpus ID: 12668798
The kinetics of hydrolysis of the antibiotic antimycin A1 has been determined. Spectrophotometric data obtained in buffer systems… Expand