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A 304000

Known as: A-304000, A304000 
National Institutes of Health

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2 relations

Broader (2)

IndolesPyridines

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2011
2011
Critical role of the transient activation of p38 MAPK in the etiology of skeletal muscle insulin resistance induced by low-level in vitro oxidant stress.
Increased cellular exposure to oxidants may contribute to the development of insulin resistance and type 2 diabetes. Skeletal… 
Highly Cited
2009
Highly Cited
2009
Oxidant stress-induced loss of IRS-1 and IRS-2 proteins in rat skeletal muscle: role of p38 MAPK.
Oxidative stress is characterized as an imbalance between the cellular production of oxidants and the cellular antioxidant… 
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Highly Cited
2009
Highly Cited
2009
Stretch‐stimulated glucose uptake in skeletal muscle is mediated by reactive oxygen species and p38 MAP‐kinase
Alternatives to the canonical insulin‐stimulated pathway for glucose uptake are exercise‐ and exogenous reactive oxygen species… 
2008
2008
Roles of insulin signalling and p38 MAPK in the activation by lithium of glucose transport in insulin-resistant rat skeletal muscle.
We have demonstrated previously in insulin-sensitive skeletal muscle that lithium, an alkali metal and non-selective inhibitor of… 
2008
2008
In vitro oxidant stress causes preferential loss of IRS proteins in rat skeletal muscle
Oxidative stress, the imbalance of oxidant production and the antioxidant defenses in the cell, can impair the functionality of… 
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2007
2007
Essential role of p38 MAPK for activation of skeletal muscle glucose transport by lithium
Abstract Lithium increases glucose transport and glycogen synthesis in insulin-sensitive cell lines and rat skeletal muscle, and… 
Highly Cited
2006
Highly Cited
2006
Oxidant stress and skeletal muscle glucose transport: roles of insulin signaling and p38 MAPK.
Oxidative stress can impact the regulation of glucose transport activity in a variety of cell lines. In the present study, we… 
2005
2005
Pharmacological inhibition of p38 MAP kinase results in improved glucose uptake in insulin-resistant 3T3-L1 adipocytes.
Inhibition of p38, a member of the mitogen-activated protein kinase family, has been shown to prevent the loss of GLUT4 protein… 
Highly Cited
2002
Highly Cited
2002
A Dominant-negative p38 MAPK Mutant and Novel Selective Inhibitors of p38 MAPK Reduce Insulin-stimulated Glucose Uptake in 3T3-L1 Adipocytes without Affecting GLUT4 Translocation*
Participation of p38 mitogen-activated protein kinase (p38) in insulin-induced glucose uptake was suggested using… 
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