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deltaFosB protein, rat

Known as: delta-FosB protein, rat 
National Institutes of Health

Papers overview

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Highly Cited
2011
Highly Cited
2011
“Snacking” causes long term attenuation of HPA axis stress responses and enhancement of brain FosB/deltaFosB expression in rats
Highly Cited
2010
Highly Cited
2010
Pharmacological recruitment of the GABAergic tail of the ventral tegmental area by acute drug exposure
BACKGROUND AND PURPOSE The tail of the ventral tegmental area (tVTA), also called the rostromedial tegmental nucleus, is a newly… 
Highly Cited
2009
Highly Cited
2009
Afferents to the GABAergic tail of the ventral tegmental area in the rat
We previously showed that chronic psychostimulant exposure induces the transcription factor DeltaFosB in γ‐aminobutyric acid… 
Highly Cited
2009
Highly Cited
2009
Ras-Guanine Nucleotide-Releasing Factor 1 (Ras-GRF1) Controls Activation of Extracellular Signal-Regulated Kinase (ERK) Signaling in the Striatum and Long-Term Behavioral Responses to Cocaine
2008
2008
Proepileptic Influence of a Focal Vascular Lesion Affecting Entorhinal Cortex-CA3 Connections After Status Epilepticus
In limbic seizures, neuronal excitation is conveyed from the entorhinal cortex directly to CA1 and subicular regions. This… 
Highly Cited
2007
Highly Cited
2007
High-Frequency Stimulation of the Subthalamic Nucleus Potentiates l-DOPA-Induced Neurochemical Changes in the Striatum in a Rat Model of Parkinson's Disease
This study examined the cellular changes produced in the striatum by chronic l-DOPA treatment and prolonged subthalamic nucleus… 
2007
2007
Pleiotropic impact of constitutive fosB inactivation on nicotine-induced behavioral alterations and stress-related traits in mice.
Multiple genes are thought to influence both susceptibility to nicotine dependence and its comorbid behavioral traits in humans… 
Highly Cited
2006
Highly Cited
2006
Effects of chronic nicotine administration and its withdrawal on striatal FosB/ΔFosB and c-Fos expression in rats and mice
2006
2006
Habituation to the test cage influences amphetamine-induced locomotion and Fos expression and increases FosB/ΔFosB-like immunoreactivity in mice
2003
2003
Chronic treatment with atypical neuroleptics induces striosomal FosB/ΔFosB expression in rats
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