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benphothiamine

Known as: BENFOTIAMINE, S-benzoylthiamine monophosphate, BTMP-benfo 
National Institutes of Health

Related topics

Related topics

5 relations

Broader (3)

Chelating AgentsImmunologic AdjuvantsThiamine
analogs & derivatives

Narrower (1)

milgamma

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
Review
2015
Review
2015
Pharmacologic Approaches Against Advanced Glycation End Products (AGEs) in Diabetic Cardiovascular Disease
Context: Advanced Glycation End-Products (AGEs) are signaling proteins associated to several vascular and neurological… 
Highly Cited
2010
Highly Cited
2010
Benfotiamine improves functional recovery of the infarcted heart via activation of pro-survival G6PD/Akt signaling pathway and modulation of neurohormonal response.
Highly Cited
2008
Highly Cited
2008
Benfotiamine exhibits direct antioxidative capacity and prevents induction of DNA damage in vitro
Complications in diabetes mellitus are partially mediated by enhanced formation of reactive oxygen species. Among the factors… 
Highly Cited
2006
Highly Cited
2006
Crystal structures of the thi-box riboswitch bound to thiamine pyrophosphate analogs reveal adaptive RNA-small molecule recognition.
Highly Cited
2006
Highly Cited
2006
Regulation of Intracellular Glucose and Polyol Pathway by Thiamine and Benfotiamine in Vascular Cells Cultured in High Glucose*
Hyperglycemia is a causal factor in the development of the vascular complications of diabetes. One of the biochemical mechanisms… 
Highly Cited
2006
Highly Cited
2006
Benfotiamine Counteracts Glucose Toxicity Effects on Endothelial Progenitor Cell Differentiation via Akt/FoxO Signaling
Dysfunction of mature endothelial cells is thought to play a major role in both micro- and macrovascular complications of… 
Highly Cited
2006
Highly Cited
2006
Benfotiamine accelerates the healing of ischaemic diabetic limbs in mice through protein kinase B/Akt-mediated potentiation of angiogenesis and inhibition of apoptosis
Aims/hypothesisBenfotiamine, a vitamin B1 analogue, reportedly prevents diabetic microangiopathy. The aim of this study was to… 
Highly Cited
2005
Highly Cited
2005
Mutations in the transketolase-like gene TKTL1: clinical implications for neurodegenerative diseases, diabetes and cancer.
Transketolase proteins or transketolase enzyme activities have been related to neurodegenerative diseases, diabetes, and cancer… 
Review
2005
Review
2005
Inhibitors of Advanced Glycation End Product Formation and Neurovascular Dysfunction in Experimental Diabetes
Abstract: Advanced glycation and lipoxidation end products (AGEs/ALEs) have been implicated in the pathogenesis of the major… 
Review
2003
Review
2003
Accumulation of fructosyl-lysine and advanced glycation end products in the kidney, retina and peripheral nerve of streptozotocin-induced diabetic rats.
The accumulation of AGEs (advanced glycation end products) in diabetes mellitus has been implicated in the biochemical… 
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