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TRDN gene
Known as:
TRDN
, TRIADIN
, TRISK
National Institutes of Health
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Related topics
Related topics
1 relation
Trdn protein, rat
Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
Review
2009
Review
2009
Junctin and the histidine‐rich Ca2+ binding protein: potential roles in heart failure and arrhythmogenesis
Tracy J. Pritchard
,
E. Kranias
Journal of Physiology
2009
Corpus ID: 20183767
Contractile dysfunction and ventricular arrhythmias associated with heart failure have been attributed to aberrant sarcoplasmic…
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Highly Cited
2008
Highly Cited
2008
Luminal Ca2+ Regulation of Single Cardiac Ryanodine Receptors: Insights Provided by Calsequestrin and its Mutants
J. Qin
,
G. Valle
,
+5 authors
M. Fill
The Journal of General Physiology
2008
Corpus ID: 7011478
The luminal Ca2+ regulation of cardiac ryanodine receptor (RyR2) was explored at the single channel level. The luminal Ca2+ and…
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Highly Cited
2007
Highly Cited
2007
On the role of junctin in cardiac Ca2+ handling, contractility, and heart failure.
U. Gergs
,
T. Berndt
,
+6 authors
J. Neumann
American Journal of Physiology. Heart and…
2007
Corpus ID: 41244974
Junctin is a transmembrane protein located at the cardiac junctional sarcoplasmic reticulum (SR) and forms a quaternary complex…
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2007
2007
Stress and high heart rate provoke ventricular tachycardia in mice expressing triadin.
P. Kirchhof
,
J. Klimas
,
+9 authors
J. Neumann
Journal of Molecular and Cellular Cardiology
2007
Corpus ID: 33947877
Highly Cited
2003
Highly Cited
2003
Junctin and calsequestrin overexpression in cardiac muscle: the role of junctin and the synthetic and delivery pathways for the two proteins.
P. Tijskens
,
L. Jones
,
C. Franzini-armstrong
Journal of Molecular and Cellular Cardiology
2003
Corpus ID: 6262037
Highly Cited
2001
Highly Cited
2001
Early impairment of calcium handling and altered expression of junctin in hearts of mice overexpressing the β1−adrenergic receptor
S. Engelhardt
,
P. Boknı́k
,
U. Keller
,
J. Neumann
,
M. Lohse
,
L. Hein
The FASEB Journal
2001
Corpus ID: 29538415
Chronic stimulation of cardiac β1‐adrenergic receptors contributes to disease progression and mortality in patients and animal…
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2001
2001
Molecular cloning and characterization of mouse cardiac triadin isoforms.
C. Hong
,
J. Ji
,
J. Kim
,
D. Jung
,
D. Kim
Gene
2001
Corpus ID: 36432015
Highly Cited
1997
Highly Cited
1997
Early functional and biochemical adaptations to low-frequency stimulation of rabbit fast-twitch muscle.
A. Hicks
,
Kay Ohlendieck
,
Sven Göpel
,
Dirk Pette
American Journal of Physiology
1997
Corpus ID: 8405378
To examine mechanisms underlying force reduction after the onset of chronic low-frequency (10 Hz) stimulation (CLFS), we exposed…
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1995
1995
Identification of triadin and of histidine-rich Ca(2+)-binding protein as substrates of 60 kDa calmodulin-dependent protein kinase in junctional terminal cisternae of sarcoplasmic reticulum of rabbit…
E. Damiani
,
E. Picello
,
Leopoldo Saggin
,
A. Margreth
Biochemical and Biophysical Research…
1995
Corpus ID: 25852429
The endogenous calmodulin-protein kinase system of sarcoplasmic reticulum terminal cisternae of rabbit fast-twitch muscle was…
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1995
1995
Localization of the N-terminal and C-terminal ends of triadin with respect to the sarcoplasmic reticulum membrane of rabbit skeletal muscle.
I. Marty
,
M. Robert
,
Michel Ronjat
,
Isabelle Bally
,
G. Arlaud
,
Michel Villaz
Biochemical Journal
1995
Corpus ID: 43404126
Antibodies were raised against synthetic peptides corresponding to the N-terminal (residues 2-17) and C-terminal (residues 691…
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