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TAZ, IVS1DS, G-C, +5

National Institutes of Health

Related topics

Related topics

1 relation
3-Methylglutaconic aciduria type 2

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2019
2019
Identification of Celastrol as a Novel YAP-TEAD Inhibitor for Cancer Therapy by High Throughput Screening with Ultrasensitive YAP/TAZ–TEAD Biosensors
The Hippo pathway has emerged as a key signaling pathway that regulates a broad range of biological functions, and dysregulation… 
2019
2019
SS18-SSX–Dependent YAP/TAZ Signaling in Synovial Sarcoma
Purpose: Synovial sarcoma is a soft tissue malignancy characterized by a reciprocal t(X;18) translocation. The chimeric SS18-SSX… 
2018
2018
Yap/Taz mediates mTORC2-stimulated fibroblast activation and kidney fibrosis
Our previously published study demonstrated that mammalian target of rapamycin complex 2 (mTORC2) signaling mediates TGFβ1… 
2016
2016
YAP/TAZ regulates the insulin signaling via IRS1/2 in endometrial cancer.
Insulin resistance (IR) is an important mechanism of pathogenesis of endometrial cancer (EC) and explains the pathogenic… 
Highly Cited
2015
Highly Cited
2015
Transcriptional co-repressor function of the hippo pathway transducers YAP and TAZ.
Highly Cited
2015
Highly Cited
2015
Crosstalk between Hippo and TGFβ: Subcellular Localization of YAP/TAZ/Smad Complexes.
Highly Cited
2015
Highly Cited
2015
A YAP/TAZ-miR-130/301 molecular circuit exerts systems-level control of fibrosis in a network of human diseases and physiologic conditions
The molecular origins of fibrosis affecting multiple tissue beds remain incompletely defined. Previously, we delineated the… 
Highly Cited
2014
Highly Cited
2014
Establishment of transgenic lines to monitor and manipulate Yap/Taz-Tead activity in zebrafish reveals both evolutionarily conserved and divergent functions of the Hippo pathway
Highly Cited
2012
Highly Cited
2012
Structural and functional similarity between the Vgll1-TEAD and the YAP-TEAD complexes.
Highly Cited
2011
Highly Cited
2011
Characterization of a transgenic short hairpin RNA-induced murine model of Tafazzin deficiency.
Barth's syndrome (BTHS) is an X-linked mitochondrial disease that is due to a mutation in the Tafazzin (TAZ) gene. Based on… 
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