SU-5416

Known as: SU 5416, SU5416, Sugen 5416 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

1957-2018
020040019572017

Papers overview

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Highly Cited
2004
Highly Cited
2004
Destruction of existing tumor blood vessels may be achieved by targeting vascular endothelial growth factor (VEGF) signaling… (More)
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Highly Cited
2003
Highly Cited
2003
Functions of receptor tyrosine kinases implicated in angiogenesis were pharmacologically impaired in a mouse model of pancreatic… (More)
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Highly Cited
2003
Highly Cited
2003
We have previously demonstrated that a failure of pulmonary endothelial cell survival induced by vascular endothelial growth… (More)
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Highly Cited
2001
Highly Cited
2001
Our understanding of the pathobiology of severe pulmonary hypertension, usually a fatal disease, has been hampered by the lack of… (More)
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Highly Cited
2000
Highly Cited
2000
During carcinogenesis of pancreatic islets in transgenic mice, an angiogenic switch activates the quiescent vasculature… (More)
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Highly Cited
2000
Highly Cited
2000
Pulmonary emphysema, a significant global health problem, is characterized by a loss of alveolar structures. Because VEGF is a… (More)
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Highly Cited
2000
Highly Cited
2000
The world volume theory on N regular and M fractional D3-branes at the conifold singularity is a non-conformal N = 1… (More)
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Highly Cited
1999
Highly Cited
1999
SU5416, a novel synthetic compound, is a potent and selective inhibitor of the Flk-1/KDR receptor tyrosine kinase that is… (More)
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Highly Cited
1999
Highly Cited
1999
Increased vascular endothelial growth factor (VEGF) expression is associated with colon cancer metastases. We hypothesized that… (More)
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Highly Cited
1996
Highly Cited
1996
The simple Bayesian classi er (SBC) is commonly thought to assume that attributes are independent given the class, but this is… (More)
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