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SP600125

National Institutes of Health

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pyrazolanthrone

Papers overview

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2015
2015
Correction: CTGF Increases IL-6 Expression in Human Synovial Fibroblasts through Integrin-Dependent Signaling Pathway
The authors would like to correct Fig 3D, as errors were introduced in the preparation of this figure for publication. In Fig 3D… 
2013
2013
Effect of SP600125 on proliferation of embryonic stem cell
SP600125 is an inhibitor of c-Jun NH2-terminal kinase (JNK), which plays a fundamental role in regulating animal development… 
2013
2013
Toll-Like Receptor-2 Ligand Peptidoglycan Upregulates Expression and Ubiquitin Ligase Activity of CHIP through JNK Pathway
Background: Peptidoglycan (PGN) is a component of cell wall in Gram-positive bacteria that stimulates inflammatory responses… 
2011
2011
Propofol Upregulates Heme Oxygenase-1 Through Activation of ERKs in Human Umbilical Vein Endothelial Cells Under Oxidative Stress Conditions
BackgroundHeme oxygenase-1 (HO-1) is an important cytoprotective agent. We examined the effect of propofol on the regulation of… 
2010
2010
Diallyl Sulfides (DAS) and Diallyl Disulfides (DADS) Exhibit a Suppressive Effect on the Proliferation and Migration of Vascular Smooth Muscle
Previous studies report that organo-sulfur compounds derived from garlic inhibited smooth muscle cell (SMC) proliferation and… 
2009
2009
Cells : The Role of JNK and Human Dermal Microvessel Endothelial Transformed Bovine Brain Endothelial Cells Lipopolysaccharide-Induced Apoptosis in
2008
2008
Pharmacological inhibitors of JNK and ERK kinases SP600125 and U0126 are not appropriate tools for studies of drug metabolism because they activate aryl hydrocarbon receptor.
Mitogen-activated protein kinases (MAPKs) are important regulators of aryl hydrocarbon receptor (AhR). An immense progress in… 
2008
2008
Nerve Growth Factor-Inducing Activity of Hericium erinaceus in 1321 N 1 Human Astrocytoma Cells
Alzheimer’s disease, for which there is currently no effective therapy, is the most common senile dementia. Alzheimer’s disease… 
2006
2006
Upregulation of TRAIL-R2 is not involved in HDACi mediated sensitization to TRAIL-induced apoptosis
Upregulation of TRAIL-R2 is not involved in HDACi mediated sensitization to TRAIL-induced apoptosis 
2005
2005
Induction of p 21 Cip 1-mediated G 2 / M arrest in H 2 O 2-treated lens epithelial cells
Reactive oxygen species (ROS), including the superoxide anion, hydrogen peroxide (H 2 O 2 ), and hydroxyl radicals are natural… 
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