SP600125

 
National Institutes of Health

Papers overview

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Highly Cited
2009
Highly Cited
2009
Our previous study showed that activation of c-jun-N-terminal kinase (JNK) in spinal astrocytes plays an important role in… (More)
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Highly Cited
2006
Highly Cited
2006
Our previous studies and the others have strongly suggested that JNK signaling pathway plays a critical role in ischemic brain… (More)
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Highly Cited
2006
Highly Cited
2006
BACKGROUND & AIMS In searching for effects of acetaminophen (APAP) on hepatocytes downstream of its metabolism that may… (More)
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Highly Cited
2004
Highly Cited
2004
Increasing evidence suggests that c-Jun N-terminal kinase (JNK) is an important kinase mediating neuronal apoptosis in Parkinson… (More)
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Highly Cited
2004
Highly Cited
2004
The c-jun N-terminal kinase (JNK) signaling pathway is regulated by JNK-interacting protein-1 (JIP1), which is a scaffolding… (More)
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Highly Cited
2004
Highly Cited
2004
After a brain insult, ATP is released from injured cells and activates microglia. The microglia that are activated in this way… (More)
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Highly Cited
2002
Highly Cited
2002
The 92-kDa type IV collagenase (MMP-9) contributes to tumor invasion and metastases and strategies to down-regulate its… (More)
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Highly Cited
2001
Highly Cited
2001
Jun N-terminal kinase (JNK) is a stress-activated protein kinase that can be induced by inflammatory cytokines, bacterial… (More)
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Highly Cited
2001
Highly Cited
2001
Mitogen-activated protein kinase (MAPK) cascades are involved in inflammation and tissue destruction in rheumatoid arthritis (RA… (More)
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Highly Cited
2001
Highly Cited
2001
Jun N-terminal kinase (JNK) is a stress-activated protein kinase that can be induced by inflammatory cytokines, bacterial… (More)
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