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SIN3B gene

Known as: KIAA0700, SIN3, YEAST, HOMOLOG OF, B, SIN3 transcription regulator family member B 
National Institutes of Health

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SIN3A gene

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2019
2019
A homozygous RNF220 mutation leads to male infertility with small-headed sperm.
2018
2018
The HDAC-Associated Sin3B Protein Represses DREAM Complex Targets and Cooperates with APC/C to Promote Quiescence
2016
2016
Negative autoregulation of BMP dependent transcription by SIN3B splicing reveals a role for RBM39
BMP signalling is negatively autoregulated by several genes including SMAD6, Noggin and Gremlin and autoregulators are possible… 
Highly Cited
2014
Highly Cited
2014
Senescence-associated SIN3B promotes inflammation and pancreatic cancer progression.
Pancreatic ductal adenocarcinoma (PDAC) is strikingly resistant to conventional therapeutic approaches. We previously… 
2011
2011
Tumor Suppressor Protein p53 Recruits Human Sin3B/HDAC1 Complex for Down-Regulation of Its Target Promoters in Response to Genotoxic Stress
Master regulator protein p53, popularly known as the “guardian of genome” is the hub for regulation of diverse cellular pathways… 
2010
2010
RNF220, an E3 ubiquitin ligase that targets Sin3B for ubiquitination.
2008
2008
Sin3B: An essential regulator of chromatin modifications at E2F target promoters during cell cycle withdrawal
Efficient and accurate cell cycle exit is intimately linked to cellular differentiation, and by inference, to the prevention of… 
2006
2006
Molecular characterization of Sin3 PAH-domain interactor specificity and identification of PAH partners
Sin3 is the central component of a multisubunit co-repressor complex. A number of DNA-binding proteins are targeted by the Sin3… 
Highly Cited
2000
Highly Cited
2000
The Mad1–Sin3B interaction involves a novel helical fold
Sin3A or Sin3B are components of a corepressor complex that mediates repression by transcription factors such as the helix-loop… 
Highly Cited
1997
Highly Cited
1997
Cell growth inhibition by the Mad/Max complex through recruitment of histone deacetylase activity
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