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SH2D1B protein, human

Known as: SH2 Domain-Containing Molecule EAT2, EWS/FLI1-Activated Transcript 2, SH2 domain containing 1B protein, human 
SH2 domain-containing protein 1B (132 aa, ~15 kDa) is encoded by the human SH2D1B gene. This protein plays a role in the regulation of receptor… 
National Institutes of Health

Related topics

Related topics

8 relations
NK Cell ActivationReceptor Activation ProcessSH2 DomainSH2D1B gene

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Papers overview

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2012
2012
Experimental validation of new drugs for calorie restriction from computational models
This work assesses a new bioinformatic tool, the Connectivity Map (Cmap), in predicting Calorie Restriction Mimetic (CRM… 
2012
2012
The chicken SLAM family
The signaling lymphocytic activation molecule (SLAM) family of receptors is critically involved in the immune regulation of… 
2011
2011
Vaccine Platforms Combining Circumsporozoite Protein and Potent Immune Modulators, rEA or EAT-2, Paradoxically Result in Opposing Immune Responses
Background Malaria greatly impacts the health and wellbeing of over half of the world's population. Promising malaria vaccine… 
2010
2010
C 57 BL / 6 Mouse CD 84-Dependent NK Cell Functions in the-2 B Are Positive Regulators of CD 244-and Cutting Edge : The Adapters EAT-2 A and
EWS/FLI1-activated transcript 2 (EAT-2)A and EAT-2B are single SH2-domain proteins, which bind to phosphorylated tyrosines of… 
2005
2005
Fine-tuning NK cell responses: it's a family affair
The adaptor SAP mediates activation signals in natural killer cells. However, its family members EAT-2 and ERT are negative… 
2005
2005
Estudi de receptors leucocitaris de la família del CD150. Identificació i caracterització dels lligands de CD84 i CD229
La familia del CD150 es troba constituida per nou proteines expressades en la superficie dels leucocits implicades en lactivacio… 
2002
2002
The SAP Family: A New Class of Adaptor-Like Molecules That Regulates Immune Cell Functions
EAT-2 is a member of a newly described adaptor-like protein family consisting, at the moment, of EAT-2 and the signaling… 
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