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Myeloid Leukemia

Known as: LEUKEMIA, GRANULOCYTIC, MALIGNANT, Myelocytic Leukemias, Unspecified myeloid leukemia 
A clonal proliferation of myeloid cells and their precursors in the bone marrow, peripheral blood, and spleen. When the proliferating cells are… 
National Institutes of Health

Papers overview

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Highly Cited
2016
Highly Cited
2016
BACKGROUND Recent studies have provided a detailed census of genes that are mutated in acute myeloid leukemia (AML). Our next… 
Highly Cited
2013
Highly Cited
2013
BACKGROUND Many mutations that contribute to the pathogenesis of acute myeloid leukemia (AML) are undefined. The relationships… 
Review
2010
Review
2010
In 2003, an international working group last reported on recommendations for diagnosis, response assessment, and treatment… 
Highly Cited
2009
Highly Cited
2009
BACKGROUND The full complement of DNA mutations that are responsible for the pathogenesis of acute myeloid leukemia (AML) is not… 
Highly Cited
2006
Highly Cited
2006
BACKGROUND The cause of chronic myeloid leukemia (CML) is a constitutively active BCR-ABL tyrosine kinase. Imatinib inhibits this… 
Highly Cited
2003
Highly Cited
2003
BACKGROUND Imatinib, a selective inhibitor of the BCR-ABL tyrosine kinase, produces high response rates in patients with chronic… 
Review
2002
Review
2002
In this chapter, Drs. Keating and Willman review recent advances in our understanding of the pathophysiology of acute myeloid… 
Highly Cited
2001
Highly Cited
2001
BACKGROUND BCR-ABL is a constitutively activated tyrosine kinase that causes chronic myeloid leukemia (CML). Since tyrosine… 
Highly Cited
2001
Highly Cited
2001
BACKGROUND BCR-ABL, a constitutively activated tyrosine kinase, is the product of the Philadelphia chromosome. This enzyme is… 
Highly Cited
1997
Highly Cited
1997
On the subject of acute myeloid leukemia (AML), there is little consensus about the target cell within the hematopoietic stem…